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Thread: More Evidence Proves Nutrition Beats Vaccines in Preventing Disease

  1. #31
    Quote Originally Posted by angelatc View Post
    This is an example of "Donna" discussing things. We have been through this about 10 times - I do not even have to click the link to respond. That isn't a "study" - it is a survey done by an anti-vax site. There is nothing random about the samples, there was no independent medical diagnosis of the ailments, etc etc etc.

    She never addresses that major point. She already knows what I am going to say, because I"ve said it before.

    And that certainly does not explain why her own source said no such studies exist while concurrently telling us to examine them..

    Most people understand that's chicanery.
    LOL! I know what you are going to say because you are predictable in your staunch advocacy for vaccines!

    That's probably what made you like Gary Johnson, he is for mandatory vaccines. I get it.
    “The spirits of darkness are now among us. We have to be on guard so that we may realize what is happening when we encounter them and gain a real idea of where they are to be found. The most dangerous thing you can do in the immediate future will be to give yourself up unconsciously to the influences which are definitely present.” ~ Rudolf Steiner



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  3. #32
    Quote Originally Posted by donnay View Post
    So you think scientific research in which pharmaceutical companies pay for has no conflict of interest?
    Do you think Big pHARMa has no influence on government in any capacity?
    Legislature forcing children in California to get vaccines is okay?

    TER we are living in deceptive times, you already have a prejudice because you are in the medical field. I understand, totally.
    No, you don't understand, because you have not taken any courses in immunology, in infectious disease, in pharmacology, in human physiology, and you do not take care of sick patients daily who come to you in trust to get the best care they can. So, frankly, you don't understand where I am coming from. Instead, you want to collectivize physicians as being under some grand delusion.

    But perhaps you have had training and I am wrong? Instead of personal online research (using selective sources and biased opinions with scant evidence based proofs), have you enrolled in any postgraduate courses to substantiate your claims of knowledge?
    Last edited by TER; 09-24-2016 at 10:55 AM.
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    'These things I command you, that you love one another.' - Jesus Christ



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  5. #33
    In reply to your questions :

    Quote Originally Posted by donnay View Post
    So you think scientific research in which pharmaceutical companies pay for has no conflict of interest?
    I never said a conflict of interest doesn't exist. I am attacking your claim that vaccines are not beneficial and that they have not saved millions of lives. Because there is corruption within the vaccination business, doesn't mean vaccines are not useful. In fact, they are, and they are important. Vaccines exist because of this reason.

    Do you think Big pHARMa has no influence on government in any capacity?
    Let me rephrase your question: "Do you think major pharmaceutical companies have no influence on government in any capacity?"

    Of course they do. It is well documented. They lobby the government. Politicians are getting rich by it.

    But that doesn't mean 'vaccines' are not beneficial and important. That only means there exists corruption in the system. Well, we know that! That's real life! Corruption is around us and in us, and of course this industry has corruption.

    But that doesn't mean 'vaccines' are not beneficial and important. They most certainly are, and thus they exist. And when someone does have the knowledge and experience, they will be able to discern that there is good, even as there may exist bad around it.

    Legislature forcing children in California to get vaccines is okay?
    This is a sociopolitical question, not a scientific question. I don't think they should make it mandatory, personally. But because I believe the government has overstepped its constitutional limits doesn't mean I don't want the vaccines for me and my family.
    Last edited by TER; 09-24-2016 at 01:10 PM.
    +
    'These things I command you, that you love one another.' - Jesus Christ

  6. #34
    As a footnote, I am not saying that all vaccines are the same or that the government should make it mandatory. My family does not take every vaccine which is offered. But there are some I will insist my children get because it can save their lives, even though it may not be 100% risk free.

    Again, we must use discernment.
    +
    'These things I command you, that you love one another.' - Jesus Christ

  7. #35
    Quote Originally Posted by TER View Post
    As a footnote, I am not saying that all vaccines are the same or that the government should make it mandatory. My family does not take every vaccine which is offered. But there are some I will insist my children get because it can save their lives, even though it may not be 100% risk free.

    Again, we must use discernment.
    Agree with the discernment. I am against vaccines but support your right to them, as long as my rights are not infringed upon.

    My cousin was stricken with polio because of a polio vaccine; a family friend's baby became deaf because of a vaccine. These were both admitted by their doctors.

    Much of my family has autistic tendencies, especially as children- my parents did a big research and feel that these were caused from vaccines. I was never vaccinated and have no autism.

    Some research:

    30 SCIENTIFIC STUDIES THAT DEMONSTRATE VAXES CAN CAUSE AUTISM

    Evidence that vaccines can cause autism

    It is an often repeated fallacy that there is no research that supports the supposition that vaccines can cause autism. This talking point is most often repeated by medical personnel and public health officials who have simply never been told that these studies exist, and in some cases by those who refuse to read the information when it is offered to them, so they continue to labor under the false assumption that vaccine autism causation is merely an “internet rumor” or a result of one paper that was published in 1998.

    This untruth was again testified to during the HHS Committee hearings


    In fact, the first research paper to offer evidence that vaccines may cause autism was THE first paper ever written on autism. In the 1930’s, Child Psychiatrist Leo Kanner discovered 11 children over the course of several years who displayed a novel set of neurological symptoms that had never been described in the medical literature, where children were withdrawn, uncommunicative and displayed similar odd behaviors. This disorder would become known as “autism.” In the paper, Dr. Kanner noted that onset of the disorder began following the administration of a small pox vaccine. This paper, was published in 1943, and evidence that vaccination causes an ever increasing rate of neurological and immunological regressions, including autism, has been mounting from that time until now.

    Autistic Disturbances of Affective Contact

    Leo Kanner, Johns Hopkins University, 1943

    “Since 1938, there have come to our attention a number of children whose condition differs so markedly and uniquely from anything reported so far, that each case merits – and, I hope, will eventually receive – at detailed consideration of its fascinating peculiarities.”

    All of Kanners cases were born after, and began to appear following, the introduction of Eli Lilly’s new form of water soluble mercury in the late 1920s used as an anti-fungal in forestry, a wood treatment product in the lumber industry and as a disinfectant and anti-bacterial in the medical industry under the name of “Thimerosal” that was included in vaccines.

    For further information on the early evidence of a vaccine/connection, I recommend reading Dr. Bryan Jepson’s book, “Changing the Course of Autism: A Scientific Approach for Parents and Physicians,” as well as Mark Blaxill and Dan Olmseted’s new book “The Age of Autism: Mercury, Medicine, and a Man-made Epidemic.”

    As I testified to at the hearing, there is abundant research supporting the vaccine autism link. I have included 49 research papers for your review, and only included research published in the last ten years or so. This is by no means a complete list, but it one that I have been compiling for the last few years as relevant research came to my attention. I have ONLY included autism related information, not research on other vaccine injuries of which there are many.

    As you can see, the medical professionals testifying that there is no scientific support for the vaccine/autism causation theory are uninformed about the current state of the science. When vaccination decisions are made based on an uninformed opinion, it means serious potential damage to the patient, and because of the law preventing lawsuits for vaccine injury, it also means that the uninformed medical professionals making bad recommendations CANNOT be held accountable in any way for giving the patient bad information.

    Parents want to know if their child can develop autism from their vaccines. If they believe that the answer is yes, and the risk of brain injury from vaccination is higher than their risk from a disease, it is their right to decline vaccination for themselves and their children with out coercion.

    Patients MUST be able to make their own informed vaccine decisions, because often, they know more about potential vaccine risks that even top public health officials do.

    1. Hepatitis B Vaccination of Male Neonates and Autism

    Annals of Epidemiology , Vol. 19, No. 9 ABSTRACTS (ACE), September 2009: 651-680,

    p. 659

    CM Gallagher, MS Goodman, Graduate Program in Public Health, Stony Brook University Medical Center, Stony Brook, NY

    PURPOSE: Universal newborn immunization with hepatitis B vaccine was recommended in 1991; however, safety findings are mixed. The Vaccine Safety Datalink Workgroup reported no association between hepatitis B vaccination at birth and febrile episodes or neurological adverse events. Other studies found positive associations between

    hepatitis B vaccination and ear infection, pharyngitis, and chronic arthritis; as well as receipt of early intervention/special education services (EIS); in probability samples of U.S. children. Children with autistic spectrum disorder (ASD) comprise a growing caseload for EIS. We evaluated the association between hepatitis B vaccination of male neonates and parental report of ASD.

    METHODS: This cross-sectional study used U.S. probability samples obtained from National Health Interview Survey 1997-2002 datasets. Logistic regression modeling was used to estimate the effect of neonatal hepatitis B vaccination on ASD risk among boys age 3-17 years with shot records, adjusted for race, maternal education, and two-parent household.

    RESULTS: Boys who received the hepatitis B vaccine during the first month of life had 2.94 greater odds for ASD (nZ31 of 7,486; OR Z 2.94; p Z 0.03; 95% CI Z 1.10, 7.90)

    compared to later- or unvaccinated boys. Non-Hispanic white boys were 61% less likely to have ASD (ORZ0.39; pZ0.04; 95% CIZ0.16, 0.94) relative to non-white boys.

    CONCLUSION: Findings suggest that U.S. male neonates vaccinated with hepatitis B vaccine had a 3-fold greater risk of ASD; risk was greatest for non-white boys.

    2. Porphyrinuria in childhood autistic disorder: Implications for environmental toxicity

    Toxicology and Applied Pharmacology, 2006

    Robert Natafa, Corinne Skorupkab, Lorene Ametb, Alain Lama, Anthea Springbettc and Richard Lathed, aLaboratoire Philippe Auguste, Paris, France, Association ARIANE, Clichy, France, Department of Statistics, Roslin Institute, Roslin, UK, Pieta Research,

    This new study from France utilizes a new and sophisticated measurement for environmental toxicity by assessing porphyrin levels in autistic children. It provides clear and unequivocal evidence that children with autism spectrum disorders are more toxic than their neurotypical peers.

    Excerpt: "Coproporphyrin levels were elevated in children with autistic disorder relative to control groups...the elevation was significant. These data implicate environmental toxicity in childhood autistic disorder."

    Abstract: To address a possible environmental contribution to autism, we carried out a retrospective study on urinary porphyrin levels, a biomarker of environmental toxicity, in 269 children with neurodevelopmental and related disorders referred to a Paris clinic (2002–2004), including 106 with autistic disorder. Urinary porphyrin levels determined by high-performance liquid chromatography were compared between diagnostic groups including internal and external control groups. Coproporphyrin levels were elevated in children with autistic disorder relative to control groups. Elevation was maintained on normalization for age or to a control heme pathway metabolite (uroporphyrin) in the same samples. The elevation was significant (P < 0.001). Porphyrin levels were unchanged in Asperger's disorder, distinguishing it from autistic disorder. The atypical molecule precoproporphyrin, a specific indicator of heavy metal toxicity, was also elevated in autistic disorder (P < 0.001) but not significantly in Asperger's. A subgroup with autistic disorder was treated with oral dimercaptosuccinic acid (DMSA) with a view to heavy metal removal. Following DMSA there was a significant (P = 0.002) drop in urinary porphyrin excretion. These data implicate environmental toxicity in childhood autistic disorder.


    3. Theoretical aspects of autism: Causes—A review


    Journal of Immunotoxicology, January-March 2011, Vol. 8, No. 1 , Pages 68-79

    Helen V. Ratajczak, PhD

    Autism, a member of the pervasive developmental disorders (PDDs), has been increasing dramatically since its description by Leo Kanner in 1943. First estimated to occur in 4 to 5 per 10,000 children, the incidence of autism is now 1 per 110 in the United States, and 1 per 64 in the United Kingdom, with similar incidences throughout the world. Searching information from 1943 to the present in PubMed and Ovid Medline databases, this review summarizes results that correlate the timing of changes in incidence with environmental changes. Autism could result from more than one cause, with different manifestations in different individuals that share common symptoms. Documented causes of autism include genetic mutations and/or deletions, viral infections, and encephalitis following vaccination. Therefore, autism is the result of genetic defects and/or inflammation of the brain. The inflammation could be caused by a defective placenta, immature blood-brain barrier, the immune response of the mother to infection while pregnant, a premature birth, encephalitis in the child after birth, or a toxic environment.

    4. Uncoupling of ATP-mediated Calcium Signaling and Dysregulated IL-6 Secretion in Dendritic Cells by Nanomolar Thimerosal

    Environmental Health Perspectives, July 2006.

    Samuel R. Goth, Ruth A. Chu Jeffrey P. Gregg

    This study demonstrates that very low-levels of Thimerosal can contribute to immune system disregulation.

    Excerpt: "Our findings that DCs primarily express the RyR1 channel complex and that this complex is uncoupled by very low levels of THI with dysregulated IL-6 secretion raise intriguing questions about a molecular basis for immune dyregulation and the possible role of the RyR1 complex in genetic susceptibility of the immune system to mercury."

    Abstract

    Dendritic cells (DCs), a rare cell type widely distributed in the soma, are potent antigen

    presenting cells that initiate primary immune responses. DCs rely on intracellular redox

    state and calcium (Ca2+) signals for proper development and function, but the relationship

    between these two signaling systems is unclear. Thimerosal (THI) is a mercurial used to

    preserve vaccines, consumer products, and experimentally to induce Ca2+ release from

    microsomal stores. We tested ATP-mediated Ca2+ responses of DCs transiently exposed

    to nanomolar THI. Transcriptional and immunocytochemical analyses show murine

    myeloid immature and mature DC (IDCs, MDCs) express inositol 1, 4, 5-trisphosphate

    and ryanodine receptor (IP3R, RyR) Ca2+ channels, known targets of THI. IDCs express

    the RyR1 isoform in a punctate distribution that is densest near plasma membranes and

    within dendritic processes whereas IP3Rs are more generally distributed. RyR1 positively

    and negatively regulates purinergic signaling since ryanodine (Ry) blockade (1) recruited

    80 percent more ATP responders, (2) shortened ATP-mediated Ca2+ transients >2-fold,

    (3) and produced a delayed and persistent rise (≥2-fold) in baseline Ca2+. THI (100nM,

    5min) recruited more ATP responders, shortened the ATP-mediated Ca2+ transient (≥1.4-

    fold) and produced a delayed rise (≥3-fold) in the Ca2+ baseline, mimicking Ry. THI and

    Ry, in combination, produced additive effects leading to uncoupling of IP3R and RyR1

    signals. THI altered ATP-mediated IL-6 secretion, initially enhancing the rate of but

    suppressing overall cytokine secretion in DCs. DCs are exquisitely sensitive to THI, with

    one mechanism involving the uncoupling of positive and negative regulation of Ca2+

    signals contributed by RyR1.

    5. Gender-selective toxicity of thimerosal.

    Exp Toxicol Pathol. 2009 Mar;61(2):133-6. Epub 2008 Sep 3.

    Branch DR, Departments of Medicine and Laboratory Medicine and Pathobiology, University of Toronto, Ontario, Canada.


    Abstract


    A recent report shows a correlation of the historical use of thimerosal in therapeutic immunizations with the subsequent development of autism; however, this association remains controversial. Autism occurs approximately four times more frequently in males compared to females; thus, studies of thimerosal toxicity should take into consideration gender-selective effects. The present study was originally undertaken to determine the maximum tolerated dose (MTD) of thimersosal in male and female CD1 mice. However, during the limited MTD studies, it became apparent that thimerosal has a differential MTD that depends on whether the mouse is male or female. At doses of 38.4-76.8mg/kg using 10% DMSO as diluent, seven of seven male mice compared to zero of seven female mice tested succumbed to thimerosal. Although the thimerosal levels used were very high, as we were originally only trying to determine MTD, it was completely unexpected to observe a difference of the MTD between male and female mice. Thus, our studies, although not directly addressing the controversy surrounding thimerosal and autism, and still preliminary due to small numbers of mice examined, provide, nevertheless, the first report of gender-selective toxicity of thimerosal and indicate that any future studies of thimerosal toxicity should take into consideration gender-specific differences.


    6. Comparison of Blood and Brain Mercury Levels in Infant Monkeys Exposed to Methylmercury or Vaccines Containing Thimerosal


    Environmental Health Perspectives, Aug 2005.

    Thomas Burbacher, PhD [University of Washington].

    This study demonstrates clearly and unequivocally that ethyl mercury, the kind of mercury found in vaccines, not only ends up in the brain, but leaves double the amount of inorganic mercury as methyl mercury, the kind of mercury found in fish. This work is groundbreaking because little is known about ethyl mercury, and many health authorities have asserted that the mercury found in vaccines is the "safe kind." This study also delivers a strong rebuke of the Institute of Medicine's recommendation in 2004 to no longer pursue the mercury-autism connection.

    Excerpt: "A recently published IOM review (IOM 2004) appears to have abandoned the earlier recommendation [of studying mercury and autism] as well as back away from the American Academy of Pediatrics goal [of removing mercury from vaccines]. This approach is difficult to understand, given our current limited knowledge of the toxicokinetics and developmental neurotoxicity of thimerosal, a compound that has been (and will continue to be) injected in millions of newborns and infants."

    7. Increases in the number of reactive glia in the visual cortex of Macaca fascicularis following subclinical long-term methyl mercury exposure.

    Toxicology and Applied Pharmacology, 1994

    Charleston JS, Bolender RP, Mottet NK, Body RL, Vahter ME, Burbacher TM., Department of Pathology, School of Medicine, University of Washington

    The number of neurons, astrocytes, reactive glia, oligodendrocytes, endothelia, and pericytes in the cortex of the calcarine sulcus of adult female Macaca fascicularis following long-term subclinical exposure to methyl mercury (MeHg) and mercuric chloride (inorganic mercury; IHg) has been estimated by use of the optical volume fractionator stereology technique. Four groups of monkeys were exposed to MeHg (50 micrograms Hg/kg body wt/day) by mouth for 6, 12, 18, and 12 months followed by 6 months without exposure (clearance group). A fifth group of monkeys was administered IHg (as HgCl2; 200 micrograms Hg/kg body wt/day) by constant rate intravenous infusion via an indwelling catheter for 3 months. Reactive glia showed a significant increase in number for every treatment group, increasing 72% in the 6-month, 152% in the 12-month, and 120% in the 18-month MeHg exposed groups, and the number of reactive glia in the clearance group remained elevated (89%). The IHg exposed group showed a 165% increase in the number of reactive glia. The IHg exposed group and the clearance group had low levels of MeHg present within the tissue; however, the level of IHg was elevated in both groups. These results suggest that the IHg may be responsible for the increase in reactive glia. All other cell types, including the neurons, showed no significant change in number at the prescribed exposure level and durations. The identities of the reactive glial cells and the implications for the long-term function and survivability of the neurons due to changes in the glial population following subclinical long-term exposure to mercury are discussed.

    8. Neuroglial Activation and Neuroinflammation in the Brain of Patients with Autism

    Annals of Neurology, Feb 2005.

    Diana L. Vargas, MD [Johns Hopkins University].

    This study, performed independently and using a different methodology than Dr. Herbert (see above) reached the same conclusion: the brains of autistic children are suffering from inflammation.

    Excerpt: "Because this neuroinflammatory process appears to be associated with an ongoing and chronic mechanism of CNS dysfunction, potential therapeutic interventions should focus on the control of its detrimental effects and thereby eventually modify the clinical course of autism."

    9. Autism: A Brain Disorder, or A Disorder That Affects the Brain?

    Clinical Neuropsychiatry, 2005

    Martha R. Herbert M.D., Ph.D., Harvard University

    Autism is defined behaviorally, as a syndrome of abnormalities involving language, social reciprocity and hyperfocus or reduced behavioral flexibility. It is clearly heterogeneous, and it can be accompanied by unusual talents as well as by impairments, but its underlying biological and genetic basis in unknown. Autism has been modeled as a brain-based, strongly genetic disorder, but emerging findings and hypotheses support a broader model of the condition as a genetically influenced and systemic. These include imaging, neuropathology and psychological evidence of pervasive (and not just specific) brain and phenotypic features; postnatal evolution and chronic persistence of brain, behavior and tissue changes (e.g. inflammation) and physical illness symptomatology (e.g. gastrointestinal, immune, recurrent infection); overlap with other disorders; and reports of rate increases and improvement or recovery that support a role for modulation of the condition by environmental factors (e.g. exacerbation or triggering by toxins, infectious agents, or others stressors, or improvement by treatment). Modeling autism more broadly encompasses previous work, but also encourages the expansion of research and treatment to include intermediary domains of molecular and cellular mechanisms, as well as chronic tissue, metabolic and somatic changes previously addressed only to a limited degree. The heterogeneous biologies underlying autism may conceivably converge onto the autism profile via multiple mechanisms on the one hand and processing and connectivity abnormalities on the other may illuminate relevant final common pathways and contribute to focusing on the search for treatment targets in this biologically and etiologically heterogeneous behavioral syndrome.


    10. Activation of Methionine Synthase by Insulin-like Growth Factor-1 and Dopamine: a Target for Neurodevelopmental Toxins and Thimerosal

    Molecular Psychiatry, July 2004.

    Richard C. Deth, PhD [Northeastern University].

    This study demonstrates how Thimerosal inhibits methylation, a central driver of cellular communication and development. Excerpt:

    "The potent inhibition of this pathway [methylation] by ethanol, lead, mercury, aluminum, and thimerosal suggests it may be an important target of neurodevelopmental toxins."

    11. Validation of the Phenomenon of Autistic Regression Using Home Videotapes

    Archives of General Psychiatry, 2005

    Emily Werner, PhD; Geraldine Dawson, PhD, University of Washington

    Objective To validate parental report of autistic regression using behavioral data coded from home videotapes of children with autism spectrum disorder (ASD) vs typical development taken at 12 and 24 months of age.

    Design Home videotapes of 56 children’s first and second birthday parties were collected from parents of young children with ASD with and without a reported history of regression and typically developing children. Child behaviors were coded by raters blind to child diagnosis and regression history. A parent interview that elicited information about parents’ recall of early symptoms from birth was also administered.

    Setting Participants were recruited from a multidisciplinary study of autism conducted at a major university.

    Participants Fifteen children with ASD with a history of regression, 21 children with ASD with early-onset autism, and 20 typically developing children and their parents participated.

    Main Outcome Measures Observations of children’s communicative, social, affective, repetitive behaviors, and toy play coded from videotapes of the toddlers’ first and second birthday parties.

    Results Analyses revealed that infants with ASD with regression show similar use of joint attention and more frequent use of words and babble compared with typical infants at 12 months of age. In contrast, infants with ASD with early onset of symptoms and no regression displayed fewer joint attention and communicative behaviors at 12 months of age. By 24 months of age, both groups of toddlers with ASD displayed fewer instances of word use, vocalizations, declarative pointing, social gaze, and orienting to name as compared with typically developing 24-month-olds.

    Parent interview data suggested that some children with regression displayed difficulties in regulatory behavior before the regression occurred.

    Conclusion This study validates the existence of early autistic regression.


    UPDATE: Since the Poling Case, this has become a popular link, so I will update it with more research and better information so that you can actually find and read the articles. Below is a partial list that I will keep adding to.

    12. Blood Levels of Mercury Are Related to Diagnosis of Autism: A Reanalysis of an Important Data Set

    Journal of Child Neurology, Vol. 22, No. 11, 1308-1311 (2007)

    M. Catherine DeSoto, PhD, Robert T. Hitlan, PhD -Department of Psychology, University of Northern Iowa, Cedar Falls, Iowa

    Excerpt: “We have reanalyzed the data set originally reported by Ip et al. in 2004 and have found that the original p value was in error and that a significant relation does exist between the blood levels of mercury and diagnosis of an autism spectrum disorder. Moreover, the hair sample analysis results offer some support for the idea that persons with autism may be less efficient and more variable at eliminating mercury from the blood.”

    Abstract

    The question of what is leading to the apparent increase in autism is of great importance. Like the link between aspirin and heart attack, even a small effect can have major health implications. If there is any link between autism and mercury, it is absolutely crucial that the first reports of the question are not falsely stating that no link occurs. We have reanalyzed the data set originally reported by Ip et al. in 2004 and have found that the original p value was in error and that a significant relation does exist between the blood levels of mercury and diagnosis of an autism spectrum disorder. Moreover, the hair sample analysis results offer some support for the idea that persons with autism may be less efficient and more variable at eliminating mercury from the blood.


    13. Developmental Regression and Mitochondrial Dysfunction in a Child With Autism


    Journal of Child Neurology / Volume 21, Number 2, February 2006

    Jon S. Poling, MD, PhD, Department of Neurology and Neurosurgery

    Johns Hopkins Hospital

    This article showed that 38% of Kennedy Krieger Institute autism patients studied had one marker for impaired oxidative phosphorylation (mitochondrial dysfunction), and 47% had a second marker.

    Excerpt: "Children who have (mitochondrial-related) dysfunctional cellular energy metabolism might be more prone to undergo autistic regression between 18 and 30 months of age if they also have infections or immunizations at the same time.”


    14. Oxidative Stress in Autism: Elevated Cerebellar 3-nitrotyrosine Levels


    Elizabeth M. Sajdel-Sulkowska, - Dept of Psychiatry, Harvard Medical School

    Shows a potential link between mercury and the autopsied brains of young people with autism. A marker for oxidative stress was 68.9% higher in autistic brain issue than controls (a statistically significant result), while mercury levels were 68.2% higher.

    Excerpt: The preliminary data suggest a need for more extensive studies of oxidative stress, its relationship to the environmental factors and its possible attenuation by antioxidants in autism.”


    15. Large Brains in Autism: The Challenge of Pervasive Abnormality


    The Neuroscientist, Volume 11, Number 5, 2005.

    Martha Herbert, MD, PhD, Harvard University

    This study helps refute the notion that the brains of autistic children are simply wired differently and notes, "neuroinflammation appears to be present in autistic brain tissue from childhood through adulthood." Dr. Herbert suggests that chronic disease or an external environmental source (like heavy metals) may be causing the inflammation.

    Excerpt: "Oxidative stress, brain inflammation, and microgliosis have been much documented in association with toxic exposures including various heavy metals...the awareness that the brain as well as medical conditions of children with autism may be conditioned by chronic biomedical abnormalities such as inflammation opens the possibility that meaningful biomedical interventions may be possible well past the window of maximal neuroplasticity in early childhood because the basis for assuming that all deficits can be attributed to fixed early developmental alterations in neural architecture has now been undermined."

    Abstract

    The most replicated finding in autism neuroanatomy—a tendency to unusually large brains—has seemed paradoxical in relation to the specificity of the abnormalities in three behavioral domains that define autism. We now know a range of things about this phenomenon, including that brains in autism have a growth spurt shortly after birth and then slow in growth a few short years afterward, that only younger but not older brains are larger in autism than in controls, that white matter contributes disproportionately to this volume increase and in a nonuniform pattern suggesting postnatal pathology, that functional connectivity among regions of autistic brains is diminished, and that neuroinflammation (including microgliosis and astrogliosis) appears to be present in autistic brain tissue from childhood through adulthood. Alongside these pervasive brain tissue and functional abnormalities, there have arisen theories of pervasive or widespread neural information processing or signal coordination abnormalities (such as weak central coherence, impaired complex processing, and underconnectivity), which are argued to underlie the specific observable behavioral features of autism. This convergence of findings and models suggests that a systems- and chronic disease–based reformulation of function and pathophysiology in autism needs to be considered, and it opens the possibility for new treatment targets.

    16. Evidence of Toxicity, Oxidative Stress, and Neuronal Insult in Autism

    Journal of Toxicology and Environmental Health, Nov-Dec 2006.

    Janet Kern, Anne Jones, Department of Psychiatry, University of Texas Southwestern Medical Center at Dallas, Dallas,

    Texas, USA

    "This article discusses the evidence for the case that some children with autism may become autistic from neuronal cell death or brain damage sometime after birth as result of insult; and addresses the hypotheses that toxicity and oxidative stress may be a cause of neuronal insult in autism... the article discusses what may be happening over the course of development and the multiple factors that may interplay and make these children more vulnerable to toxicity, oxidative stress, and neuronal insult."

    Abstract

    According to the Autism Society of America, autism is now considered to be an epidemic. The increase in the rate of autism revealed by epidemiological studies and government reports implicates the importance of external or environmental factors that may be changing. This article discusses the evidence for the case that some children with autism may become autistic from neuronal cell death or brain damage sometime after birth as result of insult; and addresses the hypotheses that toxicity and oxidative stress may be a cause of neuronal insult in autism. The article first describes the Purkinje cell loss found in

    autism, Purkinje cell physiology and vulnerability, and the evidence for postnatal cell loss. Second, the article describes the increased brain volume in autism and how it may be related to the Purkinje cell loss. Third, the evidence for toxicity and oxidative

    stress is covered and the possible involvement of glutathione is discussed. Finally, the article discusses what may be happening over the course of development and the multiple factors that may interplay and make these children more vulnerable to toxicity, oxidative stress, and neuronal insult.

    17. Oxidative Stress in Autism

    Pathophysiology, 2006.

    Abha Chauhan, Ved Chauhan

    This study provides a helpful overview of the growing evidence supporting the link between oxidative stress and autism.

    Excerpt: "Upon completion of this article, participants should be able to: 1. Be aware of laboratory and clinical evidence of greater oxidative stress in autism. 2. Understand how gut, brain, nutritional, and toxic status in autism are consistent with greater oxidative stress. 3. Describe how anti-oxidant nutrients are used in the contemporary treatment of autism."

    18. Thimerosal Neurotoxicity is Associated with Glutathione Depletion: Protection with Glutathione Precursors

    Neurotoxicology, Jan 2005.

    S. Jill James, PhD [University of Arkansas].

    This recent study demonstrates that Thimerosal lowers or inhibits the body's ability to produce Glutathione, an antioxidant and the body's primary cellular-level defense against mercury.

    Excerpt: "Thimerosal-induced cytotoxicity was associated with depletion of intracellular Glutathione in both cell lines...The potential effect of Glutathione or N-acetylcysteine against mercury toxicity warrants further research as possible adjunct therapy to individuals still receiving Thimerosal-containing vaccines."



    19. Aluminum adjuvant linked to gulf war illness induces motor neuron death in mice


    Neuromolecular Medicine, 2007

    Christopher Shaw, Ph.D. [Department of Ophthalmology and Program in Neuroscience, University of British Columbia, Vancouver, British Columbia, Canada]

    This study demonstrates the extreme toxicity of the aluminum adjuvant used as a preservative in vaccines.

    Excerpt: "testing showed motor deficits in the aluminum treatment group that expressed as a progressive decrease in strength measured...Significant cognitive deficits in water-maze learning were observed in the combined aluminum and squalene group...Apoptotic neurons were identified in aluminum-injected animals that showed significantly increased activated caspase-3 labeling in lumbar spinal cord (255%) and primary motor cortex (192%) compared with the controls. Aluminum-treated groups also showed significant motor neuron loss (35%) and increased numbers of astrocytes (350%) in the lumbar spinal cord.


    20. Environmental mercury release, special education rates, and autism disorder: an ecological study of Texas


    Health & Place, 2006

    Raymond F. Palmer, University of Texas Health Science Center

    This study demonstrated the correlation between environmental mercury and autism rates in Texas.

    Excerpt: "On average, for each 1,000 lb of environmentally released mercury, there was a 43% increase in the rate of special education services and a 61% increase in the rate of autism. The association between environmentally released mercury and special education rates were fully mediated by increased autism rates. This ecological study suggests the need for further research regarding the association between environmentally released mercury and developmental disorders such as autism."


    21. Autism Spectrum Disorders in Relation to Distribution of Hazardous Air Pollutants in the SF Bay Area


    Environmental Health Perspectives – Vol. 114 No. 9, September, 2006

    Gayle Windham, Div. of Environmental and Occupational Disease Control, California Department of Health Services

    284 ASD children & 657 controls, born in 1994 in Bay Area, were assigned exposure levels by birth tract for 19 chemicals. Risks for autism were elevated by 50% in tracts with the highest chlorinated solvents and heavy metals. The highest risk compounds were mercury, cadmium, nickel, trichloroethylene, and vinyl chloride, and the risk from heavy metals was almost twice as high as solvents.

    Excerpt: “Our results suggest a potential association between autism and estimated metal concentrations, and possibly solvents, in ambient air around the birth residence.”

    22. A Case Series of Children with Apparent Mercury Toxic Encephalopathies Manifesting with Clinical Symptoms of Regressive Autistic Disorder

    Journal of Toxicology and Environmental Health, 2007

    David A. Geier, Mark R. Geier

    This study reviewed the case histories and medical profiles of nine autistic children and concluded that eight of the nine children were mercury toxic and this toxicity manifested itself in a manner consistent with Autism Spectrum Disorders.

    Excerpt: "...these previously normally developing children suffered mercury toxic encephalopathies that manifested with clinical symptoms consistent with regressive ASDs. Evidence for mercury intoxication should be considered in the differential diagnosis as contributing to some regressive ASDs."

    Abstract

    Impairments in social relatedness and communication, repetitive behaviors, and stereotypic abnormal movement patterns characterize autism spectrum disorders (ASDs). It is clear that while genetic factors are important to the pathogenesis of ASDs, mercury exposure can induce immune, sensory, neurological, motor, and behavioral dysfunctions similar to traits defining or associated with ASDs. The Institutional Review Board of the Institute for Chronic Illnesses (Office for Human Research Protections, U.S. Department of Health and Human Services, IRB number IRB00005375) approved the present study. A case series of nine patients who presented to the Genetic Centers of America for a genetic/developmental evaluation are discussed. Eight of nine patients (one patient was found to have an ASD due to Rett’s syndrome) (a) had regressive ASDs; (b) had elevated levels of androgens; (c) excreted significant amounts of mercury post chelation challenge; (d) had biochemical evidence of decreased function in their glutathione pathways; (e) had no known significant mercury exposure except from Thimerosal- containing vaccines/Rho(D)-immune globulin preparations; and (f) had alternate causes for their regressive ASDs ruled out. There was a significant dose-response relationship between the severity of the regressive ASDs observed and the total mercury dose children received from Thimerosal-containing vaccines/Rho (D)- immune globulin preparations. Based upon differential diagnoses, 8 of 9 patients examined were exposed to significant mercury from Thimerosal-containing biologic/vaccine preparations during their fetal/infant developmental periods, and subsequently, between 12 and 24 mo of age, these previously normally developing children suffered mercury toxic encephalopathies that manifested with clinical symptoms consistent with regressive ASDs. Evidence for mercury intoxication should be considered in the differential diagnosis as contributing to some regressive ASDs.

    23. Attention-deficit hyperactivity disorder and blood mercury level: a case-control study in chinese children

    Neuropediatrics, August 2006 - P.R. Kong [Department of Pediatrics and Adolescent Medicine, The University of Hong Kong].

    This study demonstrates that blood mercury levels are higher for children with ADHD.

    Excerpt: "There was significant difference in blood mercury levels between cases and controls, which persists after adjustment for age, gender and parental occupational status. The geometric mean blood mercury level was also significantly higher in children with inattentive and combined subtypes of ADHD. High blood mercury level was associated with ADHD. Whether the relationship is causal requires further studies."

    24. The Changing Prevalence of Autism In California

    Journal of Autism and Developmental Disorders, April 2003

    Mark F. Blaxill, David S. Baskin, and Walter O. Spitzer

    This study helps to refute the supposition made by some researchers that autism's epidemic may only be due to "diagnostic substitution".

    Excerpt: "They have suggested that 'diagnostic substitution' accounts for an apparent increase in the incidence of autism in California that is not real. This hypothesized substitution is not supported by proper and detailed analyses of the California data."

    25. Mitochondrial Energy-Deficient Endophenotype in Autism

    American Journal of Biochemistry and Biotechnology 4 (2): 198-207, 2008

    J. Jay Gargus and Faiqa Imtiaz

    Department of Physiology and Biophysics and Department of Pediatrics, Section of Human Genetics, School of Medicine, University of California, Irvine, Arabian Diagnostics Laboratory, King Faisal Specialist Hospital and Research Centre

    Abstract

    While evidence points to a multigenic etiology of most autism, the pathophysiology of the disorder has yet to be defined and the underlying genes and biochemical pathways they subserve remain unknown. Autism is considered to be influenced by a combination of various genetic, environmental and immunological factors; more recently, evidence has suggested that increased vulnerability to oxidative stress may be involved in the etiology of this multifactorial disorder. Furthermore, recent studies have pointed to a subset of autism associated with the biochemical endophenotype of mitochondrial energy deficiency, identified as a subtle impairment in fat and carbohydrate oxidation. This phenotype is similar, but more subtle than those seen in classic mitochondrial defects. In some cases the beginnings of the genetic underpinnings of these mitochondrial defects are emerging, such as mild mitochondrial dysfunction and secondary carnitine deficiency observed in the subset of autistic patients with an inverted duplication of chromosome 15q11-q13. In addition, rare cases of familial autism associated with sudden infant death syndrome (SIDS) or associated with abnormalities in cellular calcium homeostasis, such as malignant hyperthermia or cardiac arrhythmia, are beginning to emerge. Such special cases suggest that the pathophysiology of autism may comprise pathways that are directly or indirectly involved in mitochondrial energy production and to further probe this connection three new avenues seem worthy of exploration: 1) metabolomic clinical studies provoking controlled aerobic exercise stress to expand the biochemical phenotype, 2) high-throughput expression arrays to directly survey activity of the genes underlying these biochemical pathways and 3) model systems, either based upon neuronal stem cells or model genetic organisms, to discover novel genetic and environmental inputs into these pathways.

    26. Bridging from Cells to Cognition in Autism Pathophysiology: Biological

    Pathways to Defective Brain Function and Plasticity


    American Journal of Biochemistry and Biotechnology 4 (2): 167-176, 2008

    Matthew P. Anderson, Brian S. Hooker and Martha R. Herbert

    Departments of Neurology and Pathology, Harvard Medical School/Beth Israel Deaconess Medical Center, Harvard Institutes of Medicine, High Throughput Biology Team, Fundamental Science Directorate, Pacific Northwest National Laboratory, Pediatric Neurology/Center for Morphometric Analysis, Massachusetts General Hospital/Harvard Medical School, and Center for Child and Adolescent Development, Cambridge Health Alliance/Harvard Medical School

    Abstract: We review evidence to support a model where the disease process underlying autism may begin when an in utero or early postnatal environmental, infectious, seizure, or autoimmune insult triggers an immune response that increases reactive oxygen species (ROS) production in the brain that leads to DNA damage (nuclear and mitochondrial) and metabolic enzyme blockade and that these inflammatory and oxidative stressors persist beyond early development (with potential further exacerbations), producing ongoing functional consequences. In organs with a high metabolic demand such as the central nervous system, the continued use of mitochondria with damaged DNA and impaired metabolic enzyme function may generate additional ROS which will cause persistent activation of the innate immune system leading to more ROS production. Such a mechanism would self-sustain and possibly progressively worsen. The mitochondrial dysfunction and altered redox signal transduction pathways found in autism would conspire to activate both astroglia and microglia. These activated cells can then initiate a broad-spectrum proinflammatory gene response. Beyond the direct effects of ROS on neuronal function, receptors on neurons that bind the inflammatory mediators may serve to inhibit neuronal signaling to protect them from excitotoxic damage during various pathologic

    insults (e.g., infection). In autism, over-zealous neuroinflammatory responses could not only influence neural developmental processes, but may more significantly impair neural signaling involved in cognition in an ongoing fashion. This model makes specific predictions in patients and experimental animal models and suggests a number of targets sites of intervention. Our model of potentially reversible pathophysiological mechanisms in autism motivates our hope that effective therapies may soon appear on the horizon.

    27. Heavy-Metal Toxicity—With Emphasis on Mercury

    John Neustadt, ND, and Steve Pieczenik, MD, PhD

    Research Review

    Conclusion: Metals are ubiquitous in our environment, and exposure to them is inevitable. However, not all people accumulate toxic levels of metals or exhibit symptoms of metal toxicity, suggesting that genetics play a role in their potential to damage health. Metal toxicity creates multisystem dysfunction, which appears to be mediated primarily through mitochondrial damage from glutathione depletion.

    Accurate screening can increase the likelihood that patients with potential metal toxicity are identified. The most accurate screening method for assessing chronic-metals exposure and metals load in the body is a provoked urine test.


    28. Evidence of Mitochondrial Dysfunction in Autism and Implications for Treatment


    American Journal of Biochemistry and Biotechnology 4 (2): 208-217, 2008

    Daniel A. Rossignol, J. Jeffrey Bradstreet, International Child Development Resource Center,

    Abstract: Classical mitochondrial diseases occur in a subset of individuals with autism and are usually caused by genetic anomalies or mitochondrial respiratory pathway deficits. However, in many cases of autism, there is evidence of mitochondrial dysfunction (MtD) without the classic features associated with mitochondrial disease. MtD appears to be more common in autism and presents with less severe signs and symptoms. It is not associated with discernable mitochondrial pathology in muscle biopsy specimens despite objective evidence of lowered mitochondrial functioning. Exposure to environmental toxins is the likely etiology for MtD in autism. This dysfunction then contributes to a number of diagnostic symptoms and comorbidities observed in autism including: cognitive impairment, language deficits, abnormal energy metabolism, chronic gastrointestinal problems, abnormalities in fatty acid oxidation, and increased oxidative stress. MtD and oxidative stress may also explain the high male to female ratio found in autism due to increased male vulnerability to these dysfunctions.

    Biomarkers for mitochondrial dysfunction have been identified, but seem widely under-utilized despite available therapeutic interventions. Nutritional supplementation to decrease oxidative stress along with factors to improve reduced glutathione, as well as hyperbaric oxygen therapy (HBOT) represent supported and rationale approaches. The underlying pathophysiology and autistic symptoms of affected individuals would be expected to either improve or cease worsening once effective treatment for MtD is implemented.


    29. Proximity to point sources of environmental mercury release as a predictor of autism prevalence

    Health & Place, 2008

    Raymond F. Palmer, Stephen Blanchard, Robert Wood

    University of Texas Health Science Center, San Antonio Department of Family and Community Medicine, Our Lady of the Lake University, San Antonio Texas, Chair, Department of Sociology

    This study should be viewed as hypothesis-generating - a first step in examining the potential role of environmental mercury and childhood developmental disorders. Nothing is known about specific exposure routes, dosage, timing, and individual susceptibility. We suspect that persistent low-dose exposures to various environmental toxicants, including mercury, that occur during critical windows of neural development among genetically susceptible children (with a diminished capacity for metabolizing accumulated toxicants) may increase the risk for developmental disorders such as autism. Successfully identifying the specific combination of environmental exposures and genetic susceptibilities can inform the development of targeted prevention intervention strategies.


    30. Epidemiology of autism spectrum disorder in Portugal: prevalence, clinical characterization, and medical conditions


    Developmental Medicine & Child Neurology, 2007

    Guiomar Oliveira MD PhD, Centro de Desenvolvimento da Criança, Hospital Pediátrico de Coimbra; Assunçăo Ataíde BSc, Direcçăo Regional de Educaçăo do Centro Coimbra;

    Carla Marques MSc, Centro de Desenvolvimento da Criança, Hospital Pediátrico de Coimbra; Teresa S Miguel BSc, Direcçăo Regional de Educaçăo do Centro, Coimbra;

    Ana Margarida Coutinho BSc, Instituto Gulbenkian de Cięncia, Oeiras; Luísa Mota-Vieira PhD, Unidade de Genética e Patologia moleculares, Hospital do Divino Espírito Santo, Ponta Delgada, Açores; Esmeralda Gonçalves PhD; Nazaré Mendes Lopes PhD, Faculdade de Cięncias e Tecnologia, Universidade de Coimbra; Vitor Rodrigues MD PhD; Henrique Carmona da Mota MD PhD, Faculdade de Medicina, Universidade de Coimbra, Coimbra; Astrid Moura Vicente PhD, Instituto Gulbenkian de Cięncia, Oeiras, Portugal.

    *Correspondence to first author at Hospital Pediátrico de Coimbra, Av Bissaya Barreto, 3000-076 Coimbra, Portugal. E-mail: guiomar@hpc.chc.min-saude.pt

    Abstract: The objective of this study was to estimate the prevalence of autistic spectrum disorder (ASD) and identify its clinical characterization, and medical conditions in a paediatric population in Portugal. A school survey was conducted in elementary schools, targeting 332 808 school-aged children in the mainland and 10 910 in the Azores islands. Referred children were directly assessed using the Diagnostic and Statistical Manual of Mental Disorders (4th edn), the Autism Diagnostic Interview–Revised, and the Childhood Autism Rating Scale. Clinical history and a laboratory investigation was performed. In parallel, a systematic multi-source search of children known to have autism was carried out in a restricted region. The global prevalence of ASD per 10 000 was 9.2 in mainland, and 15.6 in the Azores, with intriguing regional differences. A diversity of associated medical conditions was documented in 20%, with an unexpectedly high rate of mitochondrial respiratory chain disorders.[/QUOTE]
    There is no spoon.

  8. #36
    Quote Originally Posted by TER View Post
    In reply to your questions :



    I never said a conflict of interest doesn't exist. I am attacking your claim that vaccines are not beneficial and that they have not saved millions of lives. Because there is corruption within the vaccination business, doesn't mean vaccines are not useful. In fact, they are, and they are important. Vaccines exist because of this reason.



    Let me rephrase your question: "Do you think major pharmaceutical companies have no influence on government in any capacity?"

    Of course they do. It is well documented. They lobby the government. Politicians are getting rich by it.

    But that doesn't mean 'vaccines' are not beneficial and important. That only means there exists corruption in the system. Well, we know that! That's real life! Corruption is around us and in us, and of course this industry has corruption.

    But that doesn't mean 'vaccines' are not beneficial and important. They most certainly are, and thus they exist. And when someone does have the knowledge and experience, they will be able to discern that there is good, even as there may exist bad around it.



    This is a sociopolitical question, not a scientific question. I don't think they should make it mandatory, personally. But because I believe the government has overstepped its constitutional limits doesn't mean I don't want the vaccines for me and my family.
    Thanks for answering my questions but I firmly believe your answers are biased in this matter. We can just agree to disagree. If you and yours wants vaccines, I support your choice.
    Last edited by donnay; 09-24-2016 at 06:24 PM.
    “The spirits of darkness are now among us. We have to be on guard so that we may realize what is happening when we encounter them and gain a real idea of where they are to be found. The most dangerous thing you can do in the immediate future will be to give yourself up unconsciously to the influences which are definitely present.” ~ Rudolf Steiner

  9. #37
    Quote Originally Posted by donnay View Post
    Thanks for answering my questions but I firmly believe your answers are bias in this matter. We can just agree to disagree. If you and yours wants vaccines, I support your choice.
    And I believe yours is, in addition to being biased, both uninformed and without proper discernment. We can just agree to disagree and rest assure I don't believe you should be forced to agree. Just, be careful giving medical advice when you are not a medical professional. [Mod note: it is not seen where any medical advice is being given. Further, the sites Terms and Conditions state that nothing posted here should be considered medical advice.]
    +
    'These things I command you, that you love one another.' - Jesus Christ

  10. #38
    Quote Originally Posted by TER View Post
    And I believe yours is, in addition to being biased, both uninformed and without proper discernment. We can just agree to disagree and rest assure I don't believe you should be forced to agree. Just, be careful giving medical advice when you are not a medical professional.

    I do not give out medical advice.
    “The spirits of darkness are now among us. We have to be on guard so that we may realize what is happening when we encounter them and gain a real idea of where they are to be found. The most dangerous thing you can do in the immediate future will be to give yourself up unconsciously to the influences which are definitely present.” ~ Rudolf Steiner

  11. #39
    Quote Originally Posted by angelatc View Post
    It's a valid question. She does not ever discuss anything. She does not answer direct questions, she just throws up new links to move the goalposts. In other words, she's a troll.
    Lol that's rich. You, with no self awareness whatsoever, accusing Donnay of being a troll.
    I am the spoon.

  12. #40
    Quote Originally Posted by TER View Post
    No, you don't understand, because you have not taken any courses in immunology, in infectious disease, in pharmacology, in human physiology, and you do not take care of sick patients daily who come to you in trust to get the best care they can. So, frankly, you don't understand where I am coming from. Instead, you want to collectivize physicians as being under some grand delusion.

    But perhaps you have had training and I am wrong? Instead of personal online research (using selective sources and biased opinions with scant evidence based proofs), have you enrolled in any postgraduate courses to substantiate your claims of knowledge?
    You act like those courses would be taught with ZERO bias. ZERO misinformation.
    I am the spoon.



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  14. #41
    Quote Originally Posted by angelatc View Post
    This is an example of "Donna" discussing things.
    Stuff like this is why I pause when you seemingly trip over your feet to volunteer to handle certain specific duties where donations for private drives are involved around here. And the primary reason I don't like to voluntarily submit my personal information (which effectively means I can't contribute) where certain friends who eagerly volunteer to participate in those private projects or drives may access it. I'm left with questions that I don't have the answers for so can only maka reasonable judgement by ones actions (actions are a choice, btw). But you've done this before with her name. Several times. Even publicly acknowledging and distributing her first name and last initial with your little snarky and strategic quotation marks in your postings. Is it just to let her know that you know who she is and want to use it against her in order to try to intimidate her in some way? I can only imagine so by your own arbitrary actions. Why do you put her name out there in quotation marks for public consumption, angelatc? It isn't very polite. And certainly not responsible. In fact, it's malfeasant. I think that it's evil if someone made a strategic choice to do that, too. And I've called you out on it before when I've seen you do that with her name. I'm sure you remember. Is this why you do it? Hm? Is this why you always run to volunteer to do those specific things? To harvest and distribute people's personal information against them publicly if you don't like them? Hm? Ya witch? Is it? I'm just asking a question based on your own arbitrary actions.

    For instance, in one of your thread editorializations...Of course, you left the space out from in between here but elsewhere you've went out of your way to space and add your little quotation marks. I just lack the want to go looking for them. But you know I'm right.



    Anyway. Her account name is donnay. Period. Okay? That's what you refer to her by on the public board unless she expressly permits otherwise. She could temporarily take this entire site offline if she wanted to over those practices. And in a snap.

    In fact, this needs to be addressed. This is a problem. And you're getting way too comfortable with these specific antics. I don't even want to call it antics. This is something else entirely and you shouldn't, in my view, continue to be provided a platform to arbitrarily do that or otherwise remain left unchecked. I mean, really. Who do you think you are?


    From your voluntary bio, you seem rather open about feeling upset or frustrated for not having some kind of formal power/authority over others. I just wonder if you should be trusted with it. You seem very prideful and vindictive by your own demonstration and certainly demonstrate that you have no problem distributing people's real names (and who knows what else) for public consumption if it suits your personal whim. Seems kind of sociopathic.


    Political / Activist Bio:

    wHO REALLY GIVES A $#@! WHEN SPENDING HOURS BEING INVOLVED GETS YOU THE SAME RANKING AS PEOPLE WHO NEVER DO ANYTHING EXCEPT POST ON FACEBOOK
    Last edited by Natural Citizen; 09-25-2016 at 04:30 AM.

  15. #42
    Heaven forbid angelatc ever becomes a mod. Holy smoke. She'd have all of our stuff at her fingertips. And we see what she does when she knows your real name and maybe disagrees with you about something. Just puts it right out there in quotation marks for public consumption, she does. Dang. That's just evil. I don't know about you all but that gives me the heebie jeebies.

    We can't have that kind of thing going on around here. Nope. Uh uh.
    Last edited by Natural Citizen; 09-25-2016 at 04:27 AM.

  16. #43
    Quote Originally Posted by TER View Post
    Just, be careful giving medical advice when you are not a medical professional.
    Doesn't appear that she gives out any medical advice, TER. She shares articles on health related topics. This is the nature of her first amendment right so long as she is within the parameters of the rules established by the the owner of this platform as it pertains to the flow of information. Quite like when some friends sit there in the religion section talking about how much Baptists or whomever else irritate them for not obliging in some arbitrary/worldly "doctrine" or whatever.

    None the less, you've inserted an arbirary projection of the notion that there may be some consequences to her posting of health related articles. So explain, please. Why is it that she should be careful about sharing articles about health related topics? What should donnay be careful of by sharing them? Should she be afraid of something or someone for sharing health related articles, TER? Hm? If so, then what or whom, and why? Specifically, please. Thanks.

    Actually, I'm reminded of a thread I posted the other day. I'll share it here since I'm now reminded of it. To some extent anyway.

    It was about copyrights and patents in relation to corporate strategy to shut down free speech, discussion, commentary, critical analysis, speculation, and argument.

    Killing Conversation: Copyright Criminalization




    Now. Heh. Another thought on your "friendly advice" here as it pertains to "medical professionals."

    Let's talk about "medical professionals"

    I'm certain that you've heard of the Journal of the American Medical Association (JAMA) It's been the most widely circulated medical periodical in the world and one of the most respected Journal's. Right?

    Anyway, their study revealed that "medical professionals" were the third leading cause of death in United States. So I think we should be equally careful when trusting them.

    At the time of the study, these numbers were revealed to reflect deaths of hospital patients related to iatrogenic causes...and deaths only... which does not include statistics for those who survived with negative effects, btw. And I'm certain that you know what iatrogenic means.


    Deaths (year):

    12,000 -- unnecessary surgery
    7,000 -- medication errors in hospitals
    20,000 -- other errors in hospitals
    80,000 -- infections in hospitals
    106,000 -- non-error, negative effects of drugs



    Statistics for those who survived with negative effects (year):

    116 million extra physician visits
    77 million extra prescriptions
    17 million emergency department visits
    8 million hospitalizations
    3 million long-term admissions
    199,000 additional deaths
    $77 billion in extra costs


    At the time of the paper, The United States was ranked 2nd in terms of technology only to Japan. Yet when it came to success rates, it ranked toward the lower middle to bottom of industrialized nations. It was very low on the list of industrialized nations.

    So, about being careful. That's a two-way street. Wouldn't you agree?
    Last edited by Natural Citizen; 09-25-2016 at 04:51 AM.

  17. #44
    Quote Originally Posted by Ender View Post
    Agree with the discernment. I am against vaccines but support your right to them, as long as my rights are not infringed upon.

    My cousin was stricken with polio because of a polio vaccine; a family friend's baby became deaf because of a vaccine. These were both admitted by their doctors.

    Much of my family has autistic tendencies, especially as children- my parents did a big research and feel that these were caused from vaccines. I was never vaccinated and have no autism.

    Some research:

    30 SCIENTIFIC STUDIES THAT DEMONSTRATE VAXES CAN CAUSE AUTISM

    Evidence that vaccines can cause autism

    It is an often repeated fallacy that there is no research that supports the supposition that vaccines can cause autism. This talking point is most often repeated by medical personnel and public health officials who have simply never been told that these studies exist, and in some cases by those who refuse to read the information when it is offered to them, so they continue to labor under the false assumption that vaccine autism causation is merely an “internet rumor” or a result of one paper that was published in 1998.

    This untruth was again testified to during the HHS Committee hearings


    In fact, the first research paper to offer evidence that vaccines may cause autism was THE first paper ever written on autism. In the 1930’s, Child Psychiatrist Leo Kanner discovered 11 children over the course of several years who displayed a novel set of neurological symptoms that had never been described in the medical literature, where children were withdrawn, uncommunicative and displayed similar odd behaviors. This disorder would become known as “autism.” In the paper, Dr. Kanner noted that onset of the disorder began following the administration of a small pox vaccine. This paper, was published in 1943, and evidence that vaccination causes an ever increasing rate of neurological and immunological regressions, including autism, has been mounting from that time until now.

    Autistic Disturbances of Affective Contact

    Leo Kanner, Johns Hopkins University, 1943

    “Since 1938, there have come to our attention a number of children whose condition differs so markedly and uniquely from anything reported so far, that each case merits – and, I hope, will eventually receive – at detailed consideration of its fascinating peculiarities.”

    All of Kanners cases were born after, and began to appear following, the introduction of Eli Lilly’s new form of water soluble mercury in the late 1920s used as an anti-fungal in forestry, a wood treatment product in the lumber industry and as a disinfectant and anti-bacterial in the medical industry under the name of “Thimerosal” that was included in vaccines.

    For further information on the early evidence of a vaccine/connection, I recommend reading Dr. Bryan Jepson’s book, “Changing the Course of Autism: A Scientific Approach for Parents and Physicians,” as well as Mark Blaxill and Dan Olmseted’s new book “The Age of Autism: Mercury, Medicine, and a Man-made Epidemic.”

    As I testified to at the hearing, there is abundant research supporting the vaccine autism link. I have included 49 research papers for your review, and only included research published in the last ten years or so. This is by no means a complete list, but it one that I have been compiling for the last few years as relevant research came to my attention. I have ONLY included autism related information, not research on other vaccine injuries of which there are many.

    As you can see, the medical professionals testifying that there is no scientific support for the vaccine/autism causation theory are uninformed about the current state of the science. When vaccination decisions are made based on an uninformed opinion, it means serious potential damage to the patient, and because of the law preventing lawsuits for vaccine injury, it also means that the uninformed medical professionals making bad recommendations CANNOT be held accountable in any way for giving the patient bad information.

    Parents want to know if their child can develop autism from their vaccines. If they believe that the answer is yes, and the risk of brain injury from vaccination is higher than their risk from a disease, it is their right to decline vaccination for themselves and their children with out coercion.

    Patients MUST be able to make their own informed vaccine decisions, because often, they know more about potential vaccine risks that even top public health officials do.

    1. Hepatitis B Vaccination of Male Neonates and Autism

    Annals of Epidemiology , Vol. 19, No. 9 ABSTRACTS (ACE), September 2009: 651-680,

    p. 659

    CM Gallagher, MS Goodman, Graduate Program in Public Health, Stony Brook University Medical Center, Stony Brook, NY

    PURPOSE: Universal newborn immunization with hepatitis B vaccine was recommended in 1991; however, safety findings are mixed. The Vaccine Safety Datalink Workgroup reported no association between hepatitis B vaccination at birth and febrile episodes or neurological adverse events. Other studies found positive associations between

    hepatitis B vaccination and ear infection, pharyngitis, and chronic arthritis; as well as receipt of early intervention/special education services (EIS); in probability samples of U.S. children. Children with autistic spectrum disorder (ASD) comprise a growing caseload for EIS. We evaluated the association between hepatitis B vaccination of male neonates and parental report of ASD.

    METHODS: This cross-sectional study used U.S. probability samples obtained from National Health Interview Survey 1997-2002 datasets. Logistic regression modeling was used to estimate the effect of neonatal hepatitis B vaccination on ASD risk among boys age 3-17 years with shot records, adjusted for race, maternal education, and two-parent household.

    RESULTS: Boys who received the hepatitis B vaccine during the first month of life had 2.94 greater odds for ASD (nZ31 of 7,486; OR Z 2.94; p Z 0.03; 95% CI Z 1.10, 7.90)

    compared to later- or unvaccinated boys. Non-Hispanic white boys were 61% less likely to have ASD (ORZ0.39; pZ0.04; 95% CIZ0.16, 0.94) relative to non-white boys.

    CONCLUSION: Findings suggest that U.S. male neonates vaccinated with hepatitis B vaccine had a 3-fold greater risk of ASD; risk was greatest for non-white boys.

    2. Porphyrinuria in childhood autistic disorder: Implications for environmental toxicity

    Toxicology and Applied Pharmacology, 2006

    Robert Natafa, Corinne Skorupkab, Lorene Ametb, Alain Lama, Anthea Springbettc and Richard Lathed, aLaboratoire Philippe Auguste, Paris, France, Association ARIANE, Clichy, France, Department of Statistics, Roslin Institute, Roslin, UK, Pieta Research,

    This new study from France utilizes a new and sophisticated measurement for environmental toxicity by assessing porphyrin levels in autistic children. It provides clear and unequivocal evidence that children with autism spectrum disorders are more toxic than their neurotypical peers.

    Excerpt: "Coproporphyrin levels were elevated in children with autistic disorder relative to control groups...the elevation was significant. These data implicate environmental toxicity in childhood autistic disorder."

    Abstract: To address a possible environmental contribution to autism, we carried out a retrospective study on urinary porphyrin levels, a biomarker of environmental toxicity, in 269 children with neurodevelopmental and related disorders referred to a Paris clinic (2002–2004), including 106 with autistic disorder. Urinary porphyrin levels determined by high-performance liquid chromatography were compared between diagnostic groups including internal and external control groups. Coproporphyrin levels were elevated in children with autistic disorder relative to control groups. Elevation was maintained on normalization for age or to a control heme pathway metabolite (uroporphyrin) in the same samples. The elevation was significant (P < 0.001). Porphyrin levels were unchanged in Asperger's disorder, distinguishing it from autistic disorder. The atypical molecule precoproporphyrin, a specific indicator of heavy metal toxicity, was also elevated in autistic disorder (P < 0.001) but not significantly in Asperger's. A subgroup with autistic disorder was treated with oral dimercaptosuccinic acid (DMSA) with a view to heavy metal removal. Following DMSA there was a significant (P = 0.002) drop in urinary porphyrin excretion. These data implicate environmental toxicity in childhood autistic disorder.


    3. Theoretical aspects of autism: Causes—A review


    Journal of Immunotoxicology, January-March 2011, Vol. 8, No. 1 , Pages 68-79

    Helen V. Ratajczak, PhD

    Autism, a member of the pervasive developmental disorders (PDDs), has been increasing dramatically since its description by Leo Kanner in 1943. First estimated to occur in 4 to 5 per 10,000 children, the incidence of autism is now 1 per 110 in the United States, and 1 per 64 in the United Kingdom, with similar incidences throughout the world. Searching information from 1943 to the present in PubMed and Ovid Medline databases, this review summarizes results that correlate the timing of changes in incidence with environmental changes. Autism could result from more than one cause, with different manifestations in different individuals that share common symptoms. Documented causes of autism include genetic mutations and/or deletions, viral infections, and encephalitis following vaccination. Therefore, autism is the result of genetic defects and/or inflammation of the brain. The inflammation could be caused by a defective placenta, immature blood-brain barrier, the immune response of the mother to infection while pregnant, a premature birth, encephalitis in the child after birth, or a toxic environment.

    4. Uncoupling of ATP-mediated Calcium Signaling and Dysregulated IL-6 Secretion in Dendritic Cells by Nanomolar Thimerosal

    Environmental Health Perspectives, July 2006.

    Samuel R. Goth, Ruth A. Chu Jeffrey P. Gregg

    This study demonstrates that very low-levels of Thimerosal can contribute to immune system disregulation.

    Excerpt: "Our findings that DCs primarily express the RyR1 channel complex and that this complex is uncoupled by very low levels of THI with dysregulated IL-6 secretion raise intriguing questions about a molecular basis for immune dyregulation and the possible role of the RyR1 complex in genetic susceptibility of the immune system to mercury."

    Abstract

    Dendritic cells (DCs), a rare cell type widely distributed in the soma, are potent antigen

    presenting cells that initiate primary immune responses. DCs rely on intracellular redox

    state and calcium (Ca2+) signals for proper development and function, but the relationship

    between these two signaling systems is unclear. Thimerosal (THI) is a mercurial used to

    preserve vaccines, consumer products, and experimentally to induce Ca2+ release from

    microsomal stores. We tested ATP-mediated Ca2+ responses of DCs transiently exposed

    to nanomolar THI. Transcriptional and immunocytochemical analyses show murine

    myeloid immature and mature DC (IDCs, MDCs) express inositol 1, 4, 5-trisphosphate

    and ryanodine receptor (IP3R, RyR) Ca2+ channels, known targets of THI. IDCs express

    the RyR1 isoform in a punctate distribution that is densest near plasma membranes and

    within dendritic processes whereas IP3Rs are more generally distributed. RyR1 positively

    and negatively regulates purinergic signaling since ryanodine (Ry) blockade (1) recruited

    80 percent more ATP responders, (2) shortened ATP-mediated Ca2+ transients >2-fold,

    (3) and produced a delayed and persistent rise (≥2-fold) in baseline Ca2+. THI (100nM,

    5min) recruited more ATP responders, shortened the ATP-mediated Ca2+ transient (≥1.4-

    fold) and produced a delayed rise (≥3-fold) in the Ca2+ baseline, mimicking Ry. THI and

    Ry, in combination, produced additive effects leading to uncoupling of IP3R and RyR1

    signals. THI altered ATP-mediated IL-6 secretion, initially enhancing the rate of but

    suppressing overall cytokine secretion in DCs. DCs are exquisitely sensitive to THI, with

    one mechanism involving the uncoupling of positive and negative regulation of Ca2+

    signals contributed by RyR1.

    5. Gender-selective toxicity of thimerosal.

    Exp Toxicol Pathol. 2009 Mar;61(2):133-6. Epub 2008 Sep 3.

    Branch DR, Departments of Medicine and Laboratory Medicine and Pathobiology, University of Toronto, Ontario, Canada.


    Abstract


    A recent report shows a correlation of the historical use of thimerosal in therapeutic immunizations with the subsequent development of autism; however, this association remains controversial. Autism occurs approximately four times more frequently in males compared to females; thus, studies of thimerosal toxicity should take into consideration gender-selective effects. The present study was originally undertaken to determine the maximum tolerated dose (MTD) of thimersosal in male and female CD1 mice. However, during the limited MTD studies, it became apparent that thimerosal has a differential MTD that depends on whether the mouse is male or female. At doses of 38.4-76.8mg/kg using 10% DMSO as diluent, seven of seven male mice compared to zero of seven female mice tested succumbed to thimerosal. Although the thimerosal levels used were very high, as we were originally only trying to determine MTD, it was completely unexpected to observe a difference of the MTD between male and female mice. Thus, our studies, although not directly addressing the controversy surrounding thimerosal and autism, and still preliminary due to small numbers of mice examined, provide, nevertheless, the first report of gender-selective toxicity of thimerosal and indicate that any future studies of thimerosal toxicity should take into consideration gender-specific differences.


    6. Comparison of Blood and Brain Mercury Levels in Infant Monkeys Exposed to Methylmercury or Vaccines Containing Thimerosal


    Environmental Health Perspectives, Aug 2005.

    Thomas Burbacher, PhD [University of Washington].

    This study demonstrates clearly and unequivocally that ethyl mercury, the kind of mercury found in vaccines, not only ends up in the brain, but leaves double the amount of inorganic mercury as methyl mercury, the kind of mercury found in fish. This work is groundbreaking because little is known about ethyl mercury, and many health authorities have asserted that the mercury found in vaccines is the "safe kind." This study also delivers a strong rebuke of the Institute of Medicine's recommendation in 2004 to no longer pursue the mercury-autism connection.

    Excerpt: "A recently published IOM review (IOM 2004) appears to have abandoned the earlier recommendation [of studying mercury and autism] as well as back away from the American Academy of Pediatrics goal [of removing mercury from vaccines]. This approach is difficult to understand, given our current limited knowledge of the toxicokinetics and developmental neurotoxicity of thimerosal, a compound that has been (and will continue to be) injected in millions of newborns and infants."

    7. Increases in the number of reactive glia in the visual cortex of Macaca fascicularis following subclinical long-term methyl mercury exposure.

    Toxicology and Applied Pharmacology, 1994

    Charleston JS, Bolender RP, Mottet NK, Body RL, Vahter ME, Burbacher TM., Department of Pathology, School of Medicine, University of Washington

    The number of neurons, astrocytes, reactive glia, oligodendrocytes, endothelia, and pericytes in the cortex of the calcarine sulcus of adult female Macaca fascicularis following long-term subclinical exposure to methyl mercury (MeHg) and mercuric chloride (inorganic mercury; IHg) has been estimated by use of the optical volume fractionator stereology technique. Four groups of monkeys were exposed to MeHg (50 micrograms Hg/kg body wt/day) by mouth for 6, 12, 18, and 12 months followed by 6 months without exposure (clearance group). A fifth group of monkeys was administered IHg (as HgCl2; 200 micrograms Hg/kg body wt/day) by constant rate intravenous infusion via an indwelling catheter for 3 months. Reactive glia showed a significant increase in number for every treatment group, increasing 72% in the 6-month, 152% in the 12-month, and 120% in the 18-month MeHg exposed groups, and the number of reactive glia in the clearance group remained elevated (89%). The IHg exposed group showed a 165% increase in the number of reactive glia. The IHg exposed group and the clearance group had low levels of MeHg present within the tissue; however, the level of IHg was elevated in both groups. These results suggest that the IHg may be responsible for the increase in reactive glia. All other cell types, including the neurons, showed no significant change in number at the prescribed exposure level and durations. The identities of the reactive glial cells and the implications for the long-term function and survivability of the neurons due to changes in the glial population following subclinical long-term exposure to mercury are discussed.

    8. Neuroglial Activation and Neuroinflammation in the Brain of Patients with Autism

    Annals of Neurology, Feb 2005.

    Diana L. Vargas, MD [Johns Hopkins University].

    This study, performed independently and using a different methodology than Dr. Herbert (see above) reached the same conclusion: the brains of autistic children are suffering from inflammation.

    Excerpt: "Because this neuroinflammatory process appears to be associated with an ongoing and chronic mechanism of CNS dysfunction, potential therapeutic interventions should focus on the control of its detrimental effects and thereby eventually modify the clinical course of autism."

    9. Autism: A Brain Disorder, or A Disorder That Affects the Brain?

    Clinical Neuropsychiatry, 2005

    Martha R. Herbert M.D., Ph.D., Harvard University

    Autism is defined behaviorally, as a syndrome of abnormalities involving language, social reciprocity and hyperfocus or reduced behavioral flexibility. It is clearly heterogeneous, and it can be accompanied by unusual talents as well as by impairments, but its underlying biological and genetic basis in unknown. Autism has been modeled as a brain-based, strongly genetic disorder, but emerging findings and hypotheses support a broader model of the condition as a genetically influenced and systemic. These include imaging, neuropathology and psychological evidence of pervasive (and not just specific) brain and phenotypic features; postnatal evolution and chronic persistence of brain, behavior and tissue changes (e.g. inflammation) and physical illness symptomatology (e.g. gastrointestinal, immune, recurrent infection); overlap with other disorders; and reports of rate increases and improvement or recovery that support a role for modulation of the condition by environmental factors (e.g. exacerbation or triggering by toxins, infectious agents, or others stressors, or improvement by treatment). Modeling autism more broadly encompasses previous work, but also encourages the expansion of research and treatment to include intermediary domains of molecular and cellular mechanisms, as well as chronic tissue, metabolic and somatic changes previously addressed only to a limited degree. The heterogeneous biologies underlying autism may conceivably converge onto the autism profile via multiple mechanisms on the one hand and processing and connectivity abnormalities on the other may illuminate relevant final common pathways and contribute to focusing on the search for treatment targets in this biologically and etiologically heterogeneous behavioral syndrome.


    10. Activation of Methionine Synthase by Insulin-like Growth Factor-1 and Dopamine: a Target for Neurodevelopmental Toxins and Thimerosal

    Molecular Psychiatry, July 2004.

    Richard C. Deth, PhD [Northeastern University].

    This study demonstrates how Thimerosal inhibits methylation, a central driver of cellular communication and development. Excerpt:

    "The potent inhibition of this pathway [methylation] by ethanol, lead, mercury, aluminum, and thimerosal suggests it may be an important target of neurodevelopmental toxins."

    11. Validation of the Phenomenon of Autistic Regression Using Home Videotapes

    Archives of General Psychiatry, 2005

    Emily Werner, PhD; Geraldine Dawson, PhD, University of Washington

    Objective To validate parental report of autistic regression using behavioral data coded from home videotapes of children with autism spectrum disorder (ASD) vs typical development taken at 12 and 24 months of age.

    Design Home videotapes of 56 children’s first and second birthday parties were collected from parents of young children with ASD with and without a reported history of regression and typically developing children. Child behaviors were coded by raters blind to child diagnosis and regression history. A parent interview that elicited information about parents’ recall of early symptoms from birth was also administered.

    Setting Participants were recruited from a multidisciplinary study of autism conducted at a major university.

    Participants Fifteen children with ASD with a history of regression, 21 children with ASD with early-onset autism, and 20 typically developing children and their parents participated.

    Main Outcome Measures Observations of children’s communicative, social, affective, repetitive behaviors, and toy play coded from videotapes of the toddlers’ first and second birthday parties.

    Results Analyses revealed that infants with ASD with regression show similar use of joint attention and more frequent use of words and babble compared with typical infants at 12 months of age. In contrast, infants with ASD with early onset of symptoms and no regression displayed fewer joint attention and communicative behaviors at 12 months of age. By 24 months of age, both groups of toddlers with ASD displayed fewer instances of word use, vocalizations, declarative pointing, social gaze, and orienting to name as compared with typically developing 24-month-olds.

    Parent interview data suggested that some children with regression displayed difficulties in regulatory behavior before the regression occurred.

    Conclusion This study validates the existence of early autistic regression.


    UPDATE: Since the Poling Case, this has become a popular link, so I will update it with more research and better information so that you can actually find and read the articles. Below is a partial list that I will keep adding to.

    12. Blood Levels of Mercury Are Related to Diagnosis of Autism: A Reanalysis of an Important Data Set

    Journal of Child Neurology, Vol. 22, No. 11, 1308-1311 (2007)

    M. Catherine DeSoto, PhD, Robert T. Hitlan, PhD -Department of Psychology, University of Northern Iowa, Cedar Falls, Iowa

    Excerpt: “We have reanalyzed the data set originally reported by Ip et al. in 2004 and have found that the original p value was in error and that a significant relation does exist between the blood levels of mercury and diagnosis of an autism spectrum disorder. Moreover, the hair sample analysis results offer some support for the idea that persons with autism may be less efficient and more variable at eliminating mercury from the blood.”

    Abstract

    The question of what is leading to the apparent increase in autism is of great importance. Like the link between aspirin and heart attack, even a small effect can have major health implications. If there is any link between autism and mercury, it is absolutely crucial that the first reports of the question are not falsely stating that no link occurs. We have reanalyzed the data set originally reported by Ip et al. in 2004 and have found that the original p value was in error and that a significant relation does exist between the blood levels of mercury and diagnosis of an autism spectrum disorder. Moreover, the hair sample analysis results offer some support for the idea that persons with autism may be less efficient and more variable at eliminating mercury from the blood.


    13. Developmental Regression and Mitochondrial Dysfunction in a Child With Autism


    Journal of Child Neurology / Volume 21, Number 2, February 2006

    Jon S. Poling, MD, PhD, Department of Neurology and Neurosurgery

    Johns Hopkins Hospital

    This article showed that 38% of Kennedy Krieger Institute autism patients studied had one marker for impaired oxidative phosphorylation (mitochondrial dysfunction), and 47% had a second marker.

    Excerpt: "Children who have (mitochondrial-related) dysfunctional cellular energy metabolism might be more prone to undergo autistic regression between 18 and 30 months of age if they also have infections or immunizations at the same time.”


    14. Oxidative Stress in Autism: Elevated Cerebellar 3-nitrotyrosine Levels


    Elizabeth M. Sajdel-Sulkowska, - Dept of Psychiatry, Harvard Medical School

    Shows a potential link between mercury and the autopsied brains of young people with autism. A marker for oxidative stress was 68.9% higher in autistic brain issue than controls (a statistically significant result), while mercury levels were 68.2% higher.

    Excerpt: The preliminary data suggest a need for more extensive studies of oxidative stress, its relationship to the environmental factors and its possible attenuation by antioxidants in autism.”


    15. Large Brains in Autism: The Challenge of Pervasive Abnormality


    The Neuroscientist, Volume 11, Number 5, 2005.

    Martha Herbert, MD, PhD, Harvard University

    This study helps refute the notion that the brains of autistic children are simply wired differently and notes, "neuroinflammation appears to be present in autistic brain tissue from childhood through adulthood." Dr. Herbert suggests that chronic disease or an external environmental source (like heavy metals) may be causing the inflammation.

    Excerpt: "Oxidative stress, brain inflammation, and microgliosis have been much documented in association with toxic exposures including various heavy metals...the awareness that the brain as well as medical conditions of children with autism may be conditioned by chronic biomedical abnormalities such as inflammation opens the possibility that meaningful biomedical interventions may be possible well past the window of maximal neuroplasticity in early childhood because the basis for assuming that all deficits can be attributed to fixed early developmental alterations in neural architecture has now been undermined."

    Abstract

    The most replicated finding in autism neuroanatomy—a tendency to unusually large brains—has seemed paradoxical in relation to the specificity of the abnormalities in three behavioral domains that define autism. We now know a range of things about this phenomenon, including that brains in autism have a growth spurt shortly after birth and then slow in growth a few short years afterward, that only younger but not older brains are larger in autism than in controls, that white matter contributes disproportionately to this volume increase and in a nonuniform pattern suggesting postnatal pathology, that functional connectivity among regions of autistic brains is diminished, and that neuroinflammation (including microgliosis and astrogliosis) appears to be present in autistic brain tissue from childhood through adulthood. Alongside these pervasive brain tissue and functional abnormalities, there have arisen theories of pervasive or widespread neural information processing or signal coordination abnormalities (such as weak central coherence, impaired complex processing, and underconnectivity), which are argued to underlie the specific observable behavioral features of autism. This convergence of findings and models suggests that a systems- and chronic disease–based reformulation of function and pathophysiology in autism needs to be considered, and it opens the possibility for new treatment targets.

    16. Evidence of Toxicity, Oxidative Stress, and Neuronal Insult in Autism

    Journal of Toxicology and Environmental Health, Nov-Dec 2006.

    Janet Kern, Anne Jones, Department of Psychiatry, University of Texas Southwestern Medical Center at Dallas, Dallas,

    Texas, USA

    "This article discusses the evidence for the case that some children with autism may become autistic from neuronal cell death or brain damage sometime after birth as result of insult; and addresses the hypotheses that toxicity and oxidative stress may be a cause of neuronal insult in autism... the article discusses what may be happening over the course of development and the multiple factors that may interplay and make these children more vulnerable to toxicity, oxidative stress, and neuronal insult."

    Abstract

    According to the Autism Society of America, autism is now considered to be an epidemic. The increase in the rate of autism revealed by epidemiological studies and government reports implicates the importance of external or environmental factors that may be changing. This article discusses the evidence for the case that some children with autism may become autistic from neuronal cell death or brain damage sometime after birth as result of insult; and addresses the hypotheses that toxicity and oxidative stress may be a cause of neuronal insult in autism. The article first describes the Purkinje cell loss found in

    autism, Purkinje cell physiology and vulnerability, and the evidence for postnatal cell loss. Second, the article describes the increased brain volume in autism and how it may be related to the Purkinje cell loss. Third, the evidence for toxicity and oxidative

    stress is covered and the possible involvement of glutathione is discussed. Finally, the article discusses what may be happening over the course of development and the multiple factors that may interplay and make these children more vulnerable to toxicity, oxidative stress, and neuronal insult.

    17. Oxidative Stress in Autism

    Pathophysiology, 2006.

    Abha Chauhan, Ved Chauhan

    This study provides a helpful overview of the growing evidence supporting the link between oxidative stress and autism.

    Excerpt: "Upon completion of this article, participants should be able to: 1. Be aware of laboratory and clinical evidence of greater oxidative stress in autism. 2. Understand how gut, brain, nutritional, and toxic status in autism are consistent with greater oxidative stress. 3. Describe how anti-oxidant nutrients are used in the contemporary treatment of autism."

    18. Thimerosal Neurotoxicity is Associated with Glutathione Depletion: Protection with Glutathione Precursors

    Neurotoxicology, Jan 2005.

    S. Jill James, PhD [University of Arkansas].

    This recent study demonstrates that Thimerosal lowers or inhibits the body's ability to produce Glutathione, an antioxidant and the body's primary cellular-level defense against mercury.

    Excerpt: "Thimerosal-induced cytotoxicity was associated with depletion of intracellular Glutathione in both cell lines...The potential effect of Glutathione or N-acetylcysteine against mercury toxicity warrants further research as possible adjunct therapy to individuals still receiving Thimerosal-containing vaccines."



    19. Aluminum adjuvant linked to gulf war illness induces motor neuron death in mice


    Neuromolecular Medicine, 2007

    Christopher Shaw, Ph.D. [Department of Ophthalmology and Program in Neuroscience, University of British Columbia, Vancouver, British Columbia, Canada]

    This study demonstrates the extreme toxicity of the aluminum adjuvant used as a preservative in vaccines.

    Excerpt: "testing showed motor deficits in the aluminum treatment group that expressed as a progressive decrease in strength measured...Significant cognitive deficits in water-maze learning were observed in the combined aluminum and squalene group...Apoptotic neurons were identified in aluminum-injected animals that showed significantly increased activated caspase-3 labeling in lumbar spinal cord (255%) and primary motor cortex (192%) compared with the controls. Aluminum-treated groups also showed significant motor neuron loss (35%) and increased numbers of astrocytes (350%) in the lumbar spinal cord.


    20. Environmental mercury release, special education rates, and autism disorder: an ecological study of Texas


    Health & Place, 2006

    Raymond F. Palmer, University of Texas Health Science Center

    This study demonstrated the correlation between environmental mercury and autism rates in Texas.

    Excerpt: "On average, for each 1,000 lb of environmentally released mercury, there was a 43% increase in the rate of special education services and a 61% increase in the rate of autism. The association between environmentally released mercury and special education rates were fully mediated by increased autism rates. This ecological study suggests the need for further research regarding the association between environmentally released mercury and developmental disorders such as autism."


    21. Autism Spectrum Disorders in Relation to Distribution of Hazardous Air Pollutants in the SF Bay Area


    Environmental Health Perspectives – Vol. 114 No. 9, September, 2006

    Gayle Windham, Div. of Environmental and Occupational Disease Control, California Department of Health Services

    284 ASD children & 657 controls, born in 1994 in Bay Area, were assigned exposure levels by birth tract for 19 chemicals. Risks for autism were elevated by 50% in tracts with the highest chlorinated solvents and heavy metals. The highest risk compounds were mercury, cadmium, nickel, trichloroethylene, and vinyl chloride, and the risk from heavy metals was almost twice as high as solvents.

    Excerpt: “Our results suggest a potential association between autism and estimated metal concentrations, and possibly solvents, in ambient air around the birth residence.”

    22. A Case Series of Children with Apparent Mercury Toxic Encephalopathies Manifesting with Clinical Symptoms of Regressive Autistic Disorder

    Journal of Toxicology and Environmental Health, 2007

    David A. Geier, Mark R. Geier

    This study reviewed the case histories and medical profiles of nine autistic children and concluded that eight of the nine children were mercury toxic and this toxicity manifested itself in a manner consistent with Autism Spectrum Disorders.

    Excerpt: "...these previously normally developing children suffered mercury toxic encephalopathies that manifested with clinical symptoms consistent with regressive ASDs. Evidence for mercury intoxication should be considered in the differential diagnosis as contributing to some regressive ASDs."

    Abstract

    Impairments in social relatedness and communication, repetitive behaviors, and stereotypic abnormal movement patterns characterize autism spectrum disorders (ASDs). It is clear that while genetic factors are important to the pathogenesis of ASDs, mercury exposure can induce immune, sensory, neurological, motor, and behavioral dysfunctions similar to traits defining or associated with ASDs. The Institutional Review Board of the Institute for Chronic Illnesses (Office for Human Research Protections, U.S. Department of Health and Human Services, IRB number IRB00005375) approved the present study. A case series of nine patients who presented to the Genetic Centers of America for a genetic/developmental evaluation are discussed. Eight of nine patients (one patient was found to have an ASD due to Rett’s syndrome) (a) had regressive ASDs; (b) had elevated levels of androgens; (c) excreted significant amounts of mercury post chelation challenge; (d) had biochemical evidence of decreased function in their glutathione pathways; (e) had no known significant mercury exposure except from Thimerosal- containing vaccines/Rho(D)-immune globulin preparations; and (f) had alternate causes for their regressive ASDs ruled out. There was a significant dose-response relationship between the severity of the regressive ASDs observed and the total mercury dose children received from Thimerosal-containing vaccines/Rho (D)- immune globulin preparations. Based upon differential diagnoses, 8 of 9 patients examined were exposed to significant mercury from Thimerosal-containing biologic/vaccine preparations during their fetal/infant developmental periods, and subsequently, between 12 and 24 mo of age, these previously normally developing children suffered mercury toxic encephalopathies that manifested with clinical symptoms consistent with regressive ASDs. Evidence for mercury intoxication should be considered in the differential diagnosis as contributing to some regressive ASDs.

    23. Attention-deficit hyperactivity disorder and blood mercury level: a case-control study in chinese children

    Neuropediatrics, August 2006 - P.R. Kong [Department of Pediatrics and Adolescent Medicine, The University of Hong Kong].

    This study demonstrates that blood mercury levels are higher for children with ADHD.

    Excerpt: "There was significant difference in blood mercury levels between cases and controls, which persists after adjustment for age, gender and parental occupational status. The geometric mean blood mercury level was also significantly higher in children with inattentive and combined subtypes of ADHD. High blood mercury level was associated with ADHD. Whether the relationship is causal requires further studies."

    24. The Changing Prevalence of Autism In California

    Journal of Autism and Developmental Disorders, April 2003

    Mark F. Blaxill, David S. Baskin, and Walter O. Spitzer

    This study helps to refute the supposition made by some researchers that autism's epidemic may only be due to "diagnostic substitution".

    Excerpt: "They have suggested that 'diagnostic substitution' accounts for an apparent increase in the incidence of autism in California that is not real. This hypothesized substitution is not supported by proper and detailed analyses of the California data."

    25. Mitochondrial Energy-Deficient Endophenotype in Autism

    American Journal of Biochemistry and Biotechnology 4 (2): 198-207, 2008

    J. Jay Gargus and Faiqa Imtiaz

    Department of Physiology and Biophysics and Department of Pediatrics, Section of Human Genetics, School of Medicine, University of California, Irvine, Arabian Diagnostics Laboratory, King Faisal Specialist Hospital and Research Centre

    Abstract

    While evidence points to a multigenic etiology of most autism, the pathophysiology of the disorder has yet to be defined and the underlying genes and biochemical pathways they subserve remain unknown. Autism is considered to be influenced by a combination of various genetic, environmental and immunological factors; more recently, evidence has suggested that increased vulnerability to oxidative stress may be involved in the etiology of this multifactorial disorder. Furthermore, recent studies have pointed to a subset of autism associated with the biochemical endophenotype of mitochondrial energy deficiency, identified as a subtle impairment in fat and carbohydrate oxidation. This phenotype is similar, but more subtle than those seen in classic mitochondrial defects. In some cases the beginnings of the genetic underpinnings of these mitochondrial defects are emerging, such as mild mitochondrial dysfunction and secondary carnitine deficiency observed in the subset of autistic patients with an inverted duplication of chromosome 15q11-q13. In addition, rare cases of familial autism associated with sudden infant death syndrome (SIDS) or associated with abnormalities in cellular calcium homeostasis, such as malignant hyperthermia or cardiac arrhythmia, are beginning to emerge. Such special cases suggest that the pathophysiology of autism may comprise pathways that are directly or indirectly involved in mitochondrial energy production and to further probe this connection three new avenues seem worthy of exploration: 1) metabolomic clinical studies provoking controlled aerobic exercise stress to expand the biochemical phenotype, 2) high-throughput expression arrays to directly survey activity of the genes underlying these biochemical pathways and 3) model systems, either based upon neuronal stem cells or model genetic organisms, to discover novel genetic and environmental inputs into these pathways.

    26. Bridging from Cells to Cognition in Autism Pathophysiology: Biological

    Pathways to Defective Brain Function and Plasticity


    American Journal of Biochemistry and Biotechnology 4 (2): 167-176, 2008

    Matthew P. Anderson, Brian S. Hooker and Martha R. Herbert

    Departments of Neurology and Pathology, Harvard Medical School/Beth Israel Deaconess Medical Center, Harvard Institutes of Medicine, High Throughput Biology Team, Fundamental Science Directorate, Pacific Northwest National Laboratory, Pediatric Neurology/Center for Morphometric Analysis, Massachusetts General Hospital/Harvard Medical School, and Center for Child and Adolescent Development, Cambridge Health Alliance/Harvard Medical School

    Abstract: We review evidence to support a model where the disease process underlying autism may begin when an in utero or early postnatal environmental, infectious, seizure, or autoimmune insult triggers an immune response that increases reactive oxygen species (ROS) production in the brain that leads to DNA damage (nuclear and mitochondrial) and metabolic enzyme blockade and that these inflammatory and oxidative stressors persist beyond early development (with potential further exacerbations), producing ongoing functional consequences. In organs with a high metabolic demand such as the central nervous system, the continued use of mitochondria with damaged DNA and impaired metabolic enzyme function may generate additional ROS which will cause persistent activation of the innate immune system leading to more ROS production. Such a mechanism would self-sustain and possibly progressively worsen. The mitochondrial dysfunction and altered redox signal transduction pathways found in autism would conspire to activate both astroglia and microglia. These activated cells can then initiate a broad-spectrum proinflammatory gene response. Beyond the direct effects of ROS on neuronal function, receptors on neurons that bind the inflammatory mediators may serve to inhibit neuronal signaling to protect them from excitotoxic damage during various pathologic

    insults (e.g., infection). In autism, over-zealous neuroinflammatory responses could not only influence neural developmental processes, but may more significantly impair neural signaling involved in cognition in an ongoing fashion. This model makes specific predictions in patients and experimental animal models and suggests a number of targets sites of intervention. Our model of potentially reversible pathophysiological mechanisms in autism motivates our hope that effective therapies may soon appear on the horizon.

    27. Heavy-Metal Toxicity—With Emphasis on Mercury

    John Neustadt, ND, and Steve Pieczenik, MD, PhD

    Research Review

    Conclusion: Metals are ubiquitous in our environment, and exposure to them is inevitable. However, not all people accumulate toxic levels of metals or exhibit symptoms of metal toxicity, suggesting that genetics play a role in their potential to damage health. Metal toxicity creates multisystem dysfunction, which appears to be mediated primarily through mitochondrial damage from glutathione depletion.

    Accurate screening can increase the likelihood that patients with potential metal toxicity are identified. The most accurate screening method for assessing chronic-metals exposure and metals load in the body is a provoked urine test.


    28. Evidence of Mitochondrial Dysfunction in Autism and Implications for Treatment


    American Journal of Biochemistry and Biotechnology 4 (2): 208-217, 2008

    Daniel A. Rossignol, J. Jeffrey Bradstreet, International Child Development Resource Center,

    Abstract: Classical mitochondrial diseases occur in a subset of individuals with autism and are usually caused by genetic anomalies or mitochondrial respiratory pathway deficits. However, in many cases of autism, there is evidence of mitochondrial dysfunction (MtD) without the classic features associated with mitochondrial disease. MtD appears to be more common in autism and presents with less severe signs and symptoms. It is not associated with discernable mitochondrial pathology in muscle biopsy specimens despite objective evidence of lowered mitochondrial functioning. Exposure to environmental toxins is the likely etiology for MtD in autism. This dysfunction then contributes to a number of diagnostic symptoms and comorbidities observed in autism including: cognitive impairment, language deficits, abnormal energy metabolism, chronic gastrointestinal problems, abnormalities in fatty acid oxidation, and increased oxidative stress. MtD and oxidative stress may also explain the high male to female ratio found in autism due to increased male vulnerability to these dysfunctions.

    Biomarkers for mitochondrial dysfunction have been identified, but seem widely under-utilized despite available therapeutic interventions. Nutritional supplementation to decrease oxidative stress along with factors to improve reduced glutathione, as well as hyperbaric oxygen therapy (HBOT) represent supported and rationale approaches. The underlying pathophysiology and autistic symptoms of affected individuals would be expected to either improve or cease worsening once effective treatment for MtD is implemented.


    29. Proximity to point sources of environmental mercury release as a predictor of autism prevalence

    Health & Place, 2008

    Raymond F. Palmer, Stephen Blanchard, Robert Wood

    University of Texas Health Science Center, San Antonio Department of Family and Community Medicine, Our Lady of the Lake University, San Antonio Texas, Chair, Department of Sociology

    This study should be viewed as hypothesis-generating - a first step in examining the potential role of environmental mercury and childhood developmental disorders. Nothing is known about specific exposure routes, dosage, timing, and individual susceptibility. We suspect that persistent low-dose exposures to various environmental toxicants, including mercury, that occur during critical windows of neural development among genetically susceptible children (with a diminished capacity for metabolizing accumulated toxicants) may increase the risk for developmental disorders such as autism. Successfully identifying the specific combination of environmental exposures and genetic susceptibilities can inform the development of targeted prevention intervention strategies.


    30. Epidemiology of autism spectrum disorder in Portugal: prevalence, clinical characterization, and medical conditions


    Developmental Medicine & Child Neurology, 2007

    Guiomar Oliveira MD PhD, Centro de Desenvolvimento da Criança, Hospital Pediátrico de Coimbra; Assunçăo Ataíde BSc, Direcçăo Regional de Educaçăo do Centro Coimbra;

    Carla Marques MSc, Centro de Desenvolvimento da Criança, Hospital Pediátrico de Coimbra; Teresa S Miguel BSc, Direcçăo Regional de Educaçăo do Centro, Coimbra;

    Ana Margarida Coutinho BSc, Instituto Gulbenkian de Cięncia, Oeiras; Luísa Mota-Vieira PhD, Unidade de Genética e Patologia moleculares, Hospital do Divino Espírito Santo, Ponta Delgada, Açores; Esmeralda Gonçalves PhD; Nazaré Mendes Lopes PhD, Faculdade de Cięncias e Tecnologia, Universidade de Coimbra; Vitor Rodrigues MD PhD; Henrique Carmona da Mota MD PhD, Faculdade de Medicina, Universidade de Coimbra, Coimbra; Astrid Moura Vicente PhD, Instituto Gulbenkian de Cięncia, Oeiras, Portugal.

    *Correspondence to first author at Hospital Pediátrico de Coimbra, Av Bissaya Barreto, 3000-076 Coimbra, Portugal. E-mail: guiomar@hpc.chc.min-saude.pt

    Abstract: The objective of this study was to estimate the prevalence of autistic spectrum disorder (ASD) and identify its clinical characterization, and medical conditions in a paediatric population in Portugal. A school survey was conducted in elementary schools, targeting 332 808 school-aged children in the mainland and 10 910 in the Azores islands. Referred children were directly assessed using the Diagnostic and Statistical Manual of Mental Disorders (4th edn), the Autism Diagnostic Interview–Revised, and the Childhood Autism Rating Scale. Clinical history and a laboratory investigation was performed. In parallel, a systematic multi-source search of children known to have autism was carried out in a restricted region. The global prevalence of ASD per 10 000 was 9.2 in mainland, and 15.6 in the Azores, with intriguing regional differences. A diversity of associated medical conditions was documented in 20%, with an unexpectedly high rate of mitochondrial respiratory chain disorders.
    [/QUOTE]

    +Rep
    I am the spoon.

  18. #45
    Quote Originally Posted by John F Kennedy III View Post
    You act like those courses would be taught with ZERO bias. ZERO misinformation.
    I'd bet that a lot of med school entails learning how to sell various drugs these days. Depending on who is funding the material.

    Seems like there's really only 1 solution...

    Big Science: Evolution of Public Consumption
    Last edited by Natural Citizen; 09-25-2016 at 04:40 AM.

  19. #46
    Quote Originally Posted by Natural Citizen View Post
    Stuff like this is why I pause when you seemingly trip over feet to volunteer to handle duties where donations for private drives are involved around here. And the primary reason I don't like to voluntarily submit my personal information (which effectively means I can't contribute) where people who eagerly volunteer to participate in those private projects or drives may access it. I'm left with questions that I don't have the answers for so can only go by your actions. But you've done this before with her name. Several times. Even publicly acknowledging and distributing her first name and last initial with your little snarky and strategic quotation marks in your postings. Is it just to let her know that you know who she is and want to use it against her in some way in order to intimidate her in some way? I can only imagine so by your own arbitrary actions. Why do you put her name out there in quotation marks for public consumption, angelatc? I think that you're evil to do that. And I've called you out on it before when I've seen you do it. I'm sure you remember. Is this why you do it? Hm? Is this why you always run to volunteer to do those specific things? To use harvest and distribute people's personal information against them publicly if you don't like them? Hm? Ya witch? Is it? I'm just asking a question based on your arbitrary actions.

    For instance, in one of your thread editorializations...Of course, you left the space out from in between here but elsewhere you've went out of your way to space and add your little quotation marks. I just lack the want to go looking for them. But you know I'm right.



    Anyway. Her account name is donnay. Period. Okay? That's what you refer to her by on the public board unless she expressly permits otherwise. She could temporarily take this entire site offline if she wanted to over those practices. And in a snap.

    In fact, this needs to be addressed. This is a problem. And you're getting way too comfortable with these specific antics. I don't even want to call it antics. This is something else entirely and you shouldn't, in my view, continue to be provided a platform to arbitrarily do that or otherwise remain left unchecked. I mean, really. Who do you think you are?


    From your voluntary bio, you seem rather open about feeling upset or frustrated for not having some kind of formal power/authority over others. I just wonder if you should be trusted with it. You seem very prideful and vindictive by your own demonstration and certainly demonstrate that you have no problem distributing people's real names (and who knows what else) for public consumption if it suits your personal whim. Seems kind of sociopathic.
    She really should be permabanned for such actions. Doxxing someone can lead to all sorts of intended or unintended consequences.
    I am the spoon.

  20. #47
    Quote Originally Posted by Natural Citizen View Post
    I'd bet that a lot of med school entails learning how to sell various drugs these days. Depending on who is funding the material.

    Seems like there's really only 1 solution...

    Big Science: Evolution of Public Consumption
    The medical industry these days aren't much more than Big Pharma pill dispensers. Sure you can point to some good things they do, but look how much extra they charge than is even remotely necessary.
    I am the spoon.

  21. #48
    I think it's safe to say Angelatc will not even attempt to refute Ender's post and in future threads she will pretend it was never posted and she never read it.
    I am the spoon.



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  23. #49
    Quote Originally Posted by John F Kennedy III View Post
    The medical industry these days aren't much more than Big Pharma pill dispensers. Sure you can point to some good things they do, but look how much extra they charge than is even remotely necessary.
    There's no profit in cures. Only treatment. So our "medical professional" friends largely learn to treat instead of cure. And now we have this new "adherence policy" stuff that is being talked about in Washington. The pharmaceutical companies write it and the browncoats in the pharmacy police it after Dr. Feelgood prescribes it. All with federal enforcement of the policy. Pretty convenient little federalized supply and demand gig they'll have there. And defined by their own pen, no less. Cripes, we've got the biggest pharmaceutical epidemic in the world. And likely the least healthy people. That's our "medical professionals" at work there. Heh. You'd think we'd be more careful, huh?

    We've talked about the "adherence policy" stuff around here in detail. I just don't feel like looking for it to link here. It's humorous how selective the memories become when some friends profess that they've "debunked" something, though. Heh. I just kind of chuckle a bit to myself and move along to another thread when I see that.

    Mercantilists gonna mercantile.
    Last edited by Natural Citizen; 09-25-2016 at 06:12 AM.

  24. #50
    Quote Originally Posted by angelatc View Post
    (mod edit)
    Uncalled for.

    How is her being married to me in any way, shape or form, directing or protecting or biasing this argument?

    I stay away from these discussions specifically to prevent people from leveling charges of bias.

    You also assign entirely too much value to whatever "standing" I may have here.

    "Trolling" assumes that a person is taking a specious idea or belief, and running with it, in order to stir up trouble, clickbait, or draw people out into useless arguments.

    Think what want of my wife's ideas on the subject, I can tell you this: she is sincere in her beliefs and truly only wants to present info to help people.
    Last edited by Anti Federalist; 09-25-2016 at 09:24 AM.

  25. #51
    Quote Originally Posted by TER View Post
    And I believe yours is, in addition to being biased, both uninformed and without proper discernment. We can just agree to disagree and rest assure I don't believe you should be forced to agree. Just, be careful giving medical advice when you are not a medical professional.
    Also uncalled for.

    This is, in today's world, a legally actionable charge and a serious accusation. (Like what the hell is not anymore?...Three felonies a day.)

    Nowhere have I seen anything that constitutes the direct giving of medical advice.

    Flagged for removal.

  26. #52
    Quote Originally Posted by Anti Federalist View Post
    Also uncalled for.

    This is, in today's world, a legally actionable charge and a serious accusation. (Like what the hell is not anymore?...Three felonies a day.)

    Nowhere have I seen anything that constitutes the direct giving of medical advice.

    Flagged for removal.
    Promoting people to stay away from vaccines which are life saving and from conventional modern medicine on an open forum is not giving medical advice? Labeling me and all board licensed physicians as biased and under some delusional spell cast by pharmaceutical companies is not libel? I simply asked what her credentials are to promote these things and that she should be careful because, while she may have the best intentions, they may actually lead to harmful results.
    +
    'These things I command you, that you love one another.' - Jesus Christ

  27. #53
    Quote Originally Posted by TER View Post
    Promoting people to stay away from vaccines which are life saving and from conventional modern medicine on an open forum is not giving medical advice? Labeling me and all board licensed physicians as biased and under some delusional spell cast by pharmaceutical companies is not libel? I simply asked what her credentials are to promote these things and that she should be careful because, while she may have the best intentions, they may actually lead to harmful results.
    She did not give medical advice. Also following what you say could have harmful results.
    I am the spoon.

  28. #54
    Quote Originally Posted by Anti Federalist View Post
    Uncalled for.

    How is her being married to me in any way, shape or form, directing or protecting or biasing this argument?

    I stay away from these discussions specifically to prevent people from leveling charges of bias.

    You also assign entirely too much value to whatever "standing" I may have here.

    "Trolling" assumes that a person is taking a specious idea or belief, and running with it, in order to stir up trouble, clickbait, or draw people out into useless arguments.

    Think what want of my wife's ideas on the subject, I can tell you this: she is sincere in her beliefs and truly only wants to present info to help people.
    WHOA.

    I've been on this forum since 2007 & never knew you and @donnay were husband & wife? Two of my favorites on the whole blinkin' forum-

    Das Cool!
    There is no spoon.

  29. #55
    Chester Copperpot
    Member

    FWIW, I dont think either of the 2 parties arguing really thinks things out fully.

  30. #56
    Quote Originally Posted by donnay View Post


    Originally Posted by TER

    Just, be careful giving medical advice when you are not a medical professional.
    I do not give out medical advice.
    God has not given us the spirit of fear; but of power, and of love, and of a sound mind.

    Don't fall to discouragement, donnay. That is the utility of the dark side. Right? No fear...
    Last edited by Natural Citizen; 09-25-2016 at 04:16 PM.



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  32. #57
    Quote Originally Posted by Anti Federalist View Post

    Think what want of my wife's ideas on the subject, I can tell you this: she is sincere in her beliefs and truly only wants to present info to help people.

    There was a time when she'd specifically mentioned in her sig line that she was not a doctor and her news pieces/research were not intended as medical advice and that people should should seek a "medical professional."

    Probably should go back to doing that. The devil do work in mysterious ways, you know.

    Surely you see that wolves do come out in order to strike fear and to discourage.
    Last edited by Natural Citizen; 09-25-2016 at 07:04 PM.

  33. #58
    Goodbye everyone! It has been a great ride!

    I wish everyone here the best.
    +
    'These things I command you, that you love one another.' - Jesus Christ

  34. #59
    Quote Originally Posted by TER View Post
    Goodbye everyone!

    It has been a great ride!
    Later.

    I wish everyone here the best.
    Thank You. You too, TER.
    Last edited by Natural Citizen; 09-25-2016 at 04:48 PM.

  35. #60
    Quote Originally Posted by John F Kennedy III View Post
    Doxxing someone can lead to all sorts of intended or unintended consequences.
    Yep. It sure can. Speaking of doxxing, itself, since you've mentioned it, that particular tactic of harassment serves two purposes in terms of intention. 1 - It serves to intimidate and silence any targeted Individuals or groups of Individuls. And 2 - it provides an avenue for the perpetuation of that person's harassment for future harassers. In fact, I was reading a piece on that very thing and technology and security experts had mentioned that everyone from political activists to government leaders have learned how effective this attack is and do make it applicable. It's an issue of concern. Particularly if the practice is made applicable by political activists who may aquire an Individual's personal information in some way. As you may know, there are some real sociopathic cut-throats in the political activist business. Heh. Is what it is, I suppose.

    Anyway. Yeah. There are certainly intended consequences if that kind of thing is made applicable. Unintended consequences are quite another discussion.
    Last edited by Natural Citizen; 09-25-2016 at 06:58 PM.

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